Rchr
J-GLOBAL ID:200901036753751710   Update date: Feb. 01, 2024

TORU SUZUKI

スズキ トウル | TORU SUZUKI
Affiliation and department:
Research field  (2): Developmental biology ,  Molecular biology
Research keywords  (4): 肝臓、脂肪、膵臓の恒常性 ,  CCR4-NOT ,  post-transcriptional mechanism ,  分子生物学
Research theme for competitive and other funds  (6):
  • 2018 - 2021 Analysis of liver regeneration through cell-cell interaction and identification of origin of regenerated hepatocytes
  • 2009 - 2010 Analysis of the effects of cell growth and cell death regulation on tumor development using tumor mice models
  • 2007 - 2007 糖転移酵素遺伝子アレイの確立と応用
  • 2004 - 2005 細胞増殖抑制因子Tobの増殖・分化における役割とその発現制御機構の解析
  • 2003 - 2004 癌抑制遺伝子産物Tobによる細胞増殖制御機構と細胞の癌化・老化機構の解析
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Papers (42):
  • Toru Suzuki, Miyuki Hoshina, Saori Nishijima, Naosuke Hoshina, Chisato Kikuguchi, Takumi Tomohiro, Akira Fukao, Toshinobu Fujiwara, Tadashi Yamamoto. Regulation of CCR4-NOT complex deadenylase activity and cellular responses by MK2-dependent phosphorylation of CNOT2. RNA biology. 2022. 19. 1. 234-246
  • Taishin Akiyama, Toru Suzuki, Tadashi Yamamoto. RNA decay machinery safeguards immune cell development and immunological responses. Trends in immunology. 2021. 42. 5. 447-460
  • Toru Suzuki, Shungo Adachi, Chisato Kikuguchi, Shinsuke Shibata, Saori Nishijima, Yurie Kawamoto, Yusuke Iizuka, Haruhiko Koseki, Hideyuki Okano, Tohru Natsume, et al. Regulation of Fetal Genes by Transitions among RNA-Binding Proteins during Liver Development. International journal of molecular sciences. 2020. 21. 23
  • Taku Ito-Kureha, Takahisa Miyao, Saori Nishijima, Toru Suzuki, Shin-Ichi Koizumi, Alejandro Villar-Briones, Akinori Takahashi, Nobuko Akiyama, Masahiro Morita, Isao Naguro, et al. The CCR4-NOT deadenylase complex safeguards thymic positive selection by down-regulating aberrant pro-apoptotic gene expression. Nature communications. 2020. 11. 1. 6169-6169
  • Dina Mostafa, Akiko Yanagiya, Eleni Georgiadou, Yibo Wu, Theodoros Stylianides, Guy A Rutter, Toru Suzuki, Tadashi Yamamoto. Loss of β-cell identity and diabetic phenotype in mice caused by disruption of CNOT3-dependent mRNA deadenylation. Communications biology. 2020. 3. 1. 476-476
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MISC (7):
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Books (4):
  • 図説分子病態学 = Molecular patho-biochemistry and patho-biology
    中外医学社 2014 ISBN:9784498008489
  • シグナル伝達研究最前線2012 : 翻訳後修飾, 解析技術, 疾患との連関から創薬応用まで
    羊土社 2012 ISBN:9784758103213
  • 図説分子病態学
    中外医学社 2003 ISBN:4498008421
  • 細胞周期研究の新局面 : 細胞周期制御、染色体分配、減数分裂、発癌研究の最新動向とさらなる研究への広がり
    羊土社 2003 ISBN:4897060966
Education (2):
  • 1996 - 2001 The University of Tokyo
  • - 1996 The University of Tokyo Faculty of Science Department of Biophysics and Biochemistry
Professional career (1):
  • (BLANK) (The University of Tokyo)
Work history (4):
  • 2021/06 - 現在 The University of Tokyo The Institute of Medical Science
  • 2016/11 - 2022/03 理化学研究所 生命医科学研究センター・疾患遺伝研究チーム
  • 2012/04 - 2016/10 沖縄科学技術大学院大学 細胞シグナルユニット グループリーダー
  • 2001/04 - 2012/03 The University of Tokyo The Institute of Medical Science Division of Oncology
Association Membership(s) (2):
日本RNA学会 ,  日本分子生物学会
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