Research theme for competitive and other funds (5)：
2009 - 2011 アンチセンスオリゴヌクレオチドを用いた筋ジストロフィーの治療法の研究・開発
2001 - 2005 マウス神経冠細胞の感覚神経およびグリア細胞への発生運命制御
2001 - 2005 Reguratory mechanisms for cell fate choice between sensory neuron and glia
2000 - 2005 神経冠細胞の発生運命の決定に関する研究
2000 - 2005 Study on Developmental Fate of Reural Crest Cells
Papers (3)：
Mamada H, Sato T, Ota M, Sasaki H. Cell competition in mouse NIH3T3 embryonic fibroblasts is controlled by the activity of Tead family proteins and Myc. Journal of cell science. 2015
Ota M, Takeshima Y, Nishida A, Awano H, Lee T, Yagi M, Matsuo M. A G-to-T transversion at the splice acceptor site of dystrophin exon 14 shows multiple splicing outcomes that are not exemplified by transition mutations. Genetic testing and molecular biomarkers. 2012. 16. 1. 3-8
Nishida A, Kataoka N, Takeshima Y, Yagi M, Awano H, Ota M, Itoh K, Hagiwara M, Matsuo M. Chemical treatment enhances skipping of a mutated exon in the dystrophin gene. Nature communications. 2011. 2. 308
