A novel analgesic, ONO-2921, causes a use-dependent inhibition of human N-type(α<sub>1B</sub>)Ca<sup>2+</sup> channels and increases inactivated state.

SUZUKI A; OMORI H; KINOSHITA M; AKAIKE A; SATOH M; KANEKO S

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J-GLOBAL ID：200902140088049274 Reference number：02A0369356

A novel analgesic, ONO-2921, causes a use-dependent inhibition of human N-type(α_1B)Ca²⁺ channels and increases inactivated state.

新規鎮痛薬ONO-2921は用法依存性のヒトN型カルシウムチャンネルの抑制と不活性化を増強する

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Author (6)： , , , , ,
Material：
Volume： 88 Issue： Supplement 1 Page： 162 Publication year： Mar. 01, 2002
JST Material Number： G0813A ISSN： 0021-5198 CODEN： JJPAA Document type： Article
Article type：会議録記事 Country of issue： Japan (JPN) Language： ENGLISH (EN)

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Clinical neurology in general , Basic research of antipyretic analgesics

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A novel analgesic, ONO-2921, causes a use-dependent inhibition of human N-type(α1B)Ca2+ channels and increases inactivated state.

A novel analgesic, ONO-2921, causes a use-dependent inhibition of human N-type(α_1B)Ca²⁺ channels and increases inactivated state.