永田浩一

ナガタコウイチ | Nagata Koh-ichi

所属機関・部署：
職名：副所長兼部長
その他の所属（所属・部署名・職名） (1件)：

ホームページURL (1件)： http://kaken.nii.ac.jp/ja/r/50252143

研究分野 (6件)：機能生物化学 , 医化学 , 胎児医学、小児成育学 , 病態医化学 , 細胞生物学 , 脳神経外科学

研究キーワード (8件)：分子細胞生物学 , 知的障害 , 小児てんかん , 自閉性障害 , 大脳皮質発生 , 細胞骨格 , シグナル伝達 , 神経細胞

競争的資金等の研究課題 (40件)：

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論文 (165件)：

Hidenori Tabata, Koh-ichi Nagata, Kazunori Nakajima. Time-lapse imaging of migrating neurons and glial progenitors in embryonic mouse brain slices. J. Vis. Exp. 2024. 205. e66631. 1-12
Nanako Hamada, Tohru Matsuki, Ikuko Iwamoto, Takuma Nishijo, Mariko Noda, Hidenori Tabata, Atsuo Nakayama, Koh-Ichi Nagata. Expression analyses of C-terminal-binding protein 1 (CtBP1) during mouse brain development. Developmental neuroscience. 2023
Yumi Tsuneura, Taeko Kawai, Keitaro Yamada, Shintaro Aoki, Mitsuko Nakashima, Shima Eda, Tohru Matsuki, Masashi Nishikawa, Koh-ichi Nagata, Yasushi Enokido, et al. A Novel Constitutively Active $c . 98 G > C$ , p.(R33P) Variant in RAB11A Associated with Intellectual Disability Promotes Neuritogenesis and Affects Oligodendroglial Arborization. Human Mutation. 2023. 2023. 1-12
Masashi Nishikawa, Tohru Matsuki, Nanako Hamada, Atsuo Nakayama, Hidenori Ito, Koh-ichi Nagata. Expression analyses of WAC, a responsible gene for neurodevelopmental disorders, during mouse brain development. Medical Molecular Morphology. 2023
Rie Seyama, Masashi Nishikawa, Yuri Uchiyama, Keisuke Hamada, Yuka Yamamoto, Masahiro Takeda, Takanori Ochi, Monami Kishi, Toshifumi Suzuki, Kohei Hamanaka, et al. A missense variant at the RAC1-PAK1 binding site of RAC1 inactivates downstream signaling in VACTERL association. Scientific Reports. 2023. 13. 1

MISC (43件)：

講演・口頭発表等 (16件)：

Insights into the cell biology of autism through the identification and characterization of causal genes
(Symposium: Genes, Metabolites and Neural Circuits: Studying Causative Genetic Mutations to Reveal Mechanistic Pathways for Gut-Brain Nervous Development and Autism Spectrum Disorder, The 15th Meeting of the Asian-Pacific Society for Neurochemistry 2018)
Comprehensive analyses of causative genes for ASD with in utero electropolation technique
(International Symposium on Autism 2017 2017)
Comprehensive analyses of RBFOX1, a causative gene for neurodevelopmental and psychiatric diseases, in the brain development
(Symposium: Common molecular aspects of neurodevelopmental and psychiatric disorders. The 14th Meeting of the Asian-Pacific Society for Neurochemistry 2016)
Comprehensive approach to understand pathophysiological role of SIL1, a gene causing intellectual disability
(Symposium; Harnessing Human Genetics to Define the Biochemical Pathways involved in Brain Development: 25th Meeting of International Society for Neurochemistry 2015)
Comprehensive approach with an analytical battery to understand pathophysiological role of A2BP1/RBFOX1, a "hub" gene in the ASD gene transcriptome network
(Keystone Symposia on Molecular and Cellular Biology Pathways of Neurodevelopmental Disorders 2015)

学歴 (2件)：

学位 (1件)：

経歴 (6件)：

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委員歴 (4件)：

受賞 (3件)：

所属学会 (7件)：

米国神経化学会 , 日本小児神経学会 , 米国細胞生物学会 , 国際神経化学会 , 日本神経化学会 , 日本臨床分子形態学会 , 日本生化学会

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永田 浩一