文献
J-GLOBAL ID:201702224771149437
整理番号:17A1826399
CCL19/CCR7はES細胞の増殖と浸潤を調節することによりPI3K/Akt経路を介して子宮内膜症の病因に寄与する【Powered by NICT】
CCL19/CCR7 contributes to the pathogenesis of endometriosis via PI3K/Akt pathway by regulating the proliferation and invasion of ESCs
著者 (7件):
Diao Ruiying
(Centre of Reproductive Medicine, Shenzhen Second People’s Hospital, The First Affiliated Hospital of Shenzhen University, Shenzhen, China)
,
Wei Weixia
(Department of Obstetrics and Gynecology, Peking University Shenzhen Hospital, Shenzhen, China)
,
Zhao Jinghui
(Department of Obstetrics and Gynecology, Peking University Shenzhen Hospital, Shenzhen, China)
,
Zhao Jinghui
(Reproductive Medicine Centre, Children’s Hospital of Shanxi Province, Shanxi, China)
,
Tian Fuying
(Centre of Reproductive Medicine, Shenzhen Second People’s Hospital, The First Affiliated Hospital of Shenzhen University, Shenzhen, China)
,
Cai Xueyong
(Centre of Reproductive Medicine, Shenzhen Second People’s Hospital, The First Affiliated Hospital of Shenzhen University, Shenzhen, China)
,
Duan Yong-Gang
(Shenzhen Key Laboratory of Fertility Regulation, Center of Assisted Reproduction and Embryology, The University of Hong Kong - Shenzhen Hospital, Shenzhen, China)
資料名:
American Journal of Reproductive Immunology
(American Journal of Reproductive Immunology)
巻:
78
号:
5
ページ:
ROMBUNNO.12744
発行年:
2017年
JST資料番号:
W2490A
ISSN:
1046-7408
資料種別:
逐次刊行物 (A)
記事区分:
原著論文
発行国:
アメリカ合衆国 (USA)
言語:
英語 (EN)