研究者
J-GLOBAL ID:201801001188491124   更新日: 2024年01月30日

喜名 美香

キナ ミカ | Kina Mika
所属機関・部署:
研究分野 (1件): 外科系歯学
競争的資金等の研究課題 (5件):
  • 2021 - 2024 EphA4活性化により生じる高分化型口腔癌メトロノーム化学療法耐性機構の解明
  • 2019 - 2023 内臓脂肪型肥満発症経路が関与する抗癌剤耐性機構の解明
  • 2018 - 2021 抗癌剤耐性高分化型口腔癌に対するEphA4を標的とした分子生学的メカニズムの解明
  • 2017 - 2020 抗癌剤曝露依存的に活性化されるEphA4 を標的とした新規治療戦略の構築
  • 2015 - 2017 食事中の亜硝酸/硝酸の不足にて生じる代謝症候群の分子機構の解明と新規治療法の開発
論文 (5件):
  • Mika Kina-Tanada, Mayuko Sakanashi, Akira Arasaki, Masato Tsutsui. [Long-term dietary nitrite and nitrate deficiency causes metabolic syndrome, endothelial dysfunction, and cardiovascular death in mice]. Nihon yakurigaku zasshi. Folia pharmacologica Japonica. 2018. 151. 4. 148-154
  • Mika Kina-Tanada, Mayuko Sakanashi, Akihide Tanimoto, Tadashi Kaname, Toshihiro Matsuzaki, Katsuhiko Noguchi, Taro Uchida, Junko Nakasone, Chisayo Kozuka, Masayoshi Ishida, et al. Long-term dietary nitrite and nitrate deficiency causes the metabolic syndrome, endothelial dysfunction and cardiovascular death in mice. Diabetologia. 2017. 60. 6. 1138-1151
  • Katsuhiko Noguchi, Toshihiro Matsuzaki, Mayuko Sakanashi, Naobumi Hamadate, Taro Uchida, Mika Kina-Tanada, Haruaki Kubota, Junko Nakasone, Matao Sakanashi, Shinichiro Ueda, et al. Effect of caffeine contained in a cup of coffee on microvascular function in healthy subjects. Journal of pharmacological sciences. 2015. 127. 2. 217-22
  • Taro Uchida, Yumi Furuno, Akihide Tanimoto, Yumiko Toyohira, Kumiko Arakaki, Mika Kina-Tanada, Haruaki Kubota, Mayuko Sakanashi, Toshihiro Matsuzaki, Katsuhiko Noguchi, et al. Development of an experimentally useful model of acute myocardial infarction: 2/3 nephrectomized triple nitric oxide synthases-deficient mouse. Journal of molecular and cellular cardiology. 2014. 77. 29-41
  • Mayuko Sakanashi, Toshihiro Matsuzaki, Katsuhiko Noguchi, Junko Nakasone, Makiko Sakanashi, Taro Uchida, Mika Kina-Tanada, Haruaki Kubota, Kumiko Arakaki, Akihide Tanimoto, et al. Long-term treatment with san'o-shashin-to, a kampo medicine, markedly ameliorates cardiac ischemia-reperfusion injury in ovariectomized rats via the redox-dependent mechanism. Circulation journal : official journal of the Japanese Circulation Society. 2013. 77. 7. 1827-37
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