研究者
J-GLOBAL ID：202001015563693332   更新日: 2024年02月01日

齋藤 晶理

サイトウ アキミチ | Saito Akimichi
所属機関・部署：
北海道医療大学 予防医療科学センター

北海道医療大学 予防医療科学センター について


職名： 准教授
競争的資金等の研究課題 (3件)：
  • 2018 - 2020 心筋炎におけるナチュラルキラーT細胞の役割の解明
  • 2012 - 2015 心筋リモデリングにおけるナチュラルキラーT細胞による炎症制御機構の解明と治療応用
  • 2011 - 2015 ナチュラルキラーT細胞による動脈硬化における慢性炎症遷延化の分子基盤の解明
論文 (13件)：
  • Yoshinori Ohmura, Naoki Ishimori, Akimichi Saito, Takashi Yokota, Shunpei Horii, Satoshi Tokuhara, Kazuya Iwabuchi, Hiroyuki Tsutsui. Natural Killer T Cells Are Involved in Atherosclerotic Plaque Instability in Apolipoprotein-E Knockout Mice. International journal of molecular sciences. 2021. 22. 22
  • Akimichi Saito, Naoki Ishimori, Satoshi Tokuhara, Tsuneaki Homma, Mikito Nishikawa, Kazuya Iwabuchi, Hiroyuki Tsutsui. Activation of Invariant Natural Killer T Cells by α-Galactosylceramide Attenuates the Development of Angiotensin II-Mediated Abdominal Aortic Aneurysm in Obese ob/ob Mice. Frontiers in Cardiovascular Medicine. 2021. 8
  • Hideo Nambu, Shingo Takada, Satoshi Maekawa, Junichi Matsumoto, Naoya Kakutani, Takaaki Furihata, Ryosuke Shirakawa, Takashi Katayama, Takayuki Nakajima, Katsuma Yamanashi, et al. Inhibition of xanthine oxidase in the acute phase of myocardial infarction prevents skeletal muscle abnormalities and exercise intolerance. Cardiovascular research. 2021. 117. 3. 805-819
  • Yoshikuni Obata, Naoki Ishimori, Akimichi Saito, Shintaro Kinugawa, Takashi Yokota, Shingo Takada, Ippei Nakano, Naoya Kakutani, Katsuma Yamanashi, Toshihisa Anzai. Activation of invariant natural killer T cells by alpha-galactosylceramide ameliorates doxorubicin-induced cardiotoxicity in mice. European journal of preventive cardiology. 2020. 27. 19. 2047487319901208-2047487319901208
  • Masashige Takahashi, Shintaro Kinugawa, Shingo Takada, Naoya Kakutani, Takaaki Furihata, Mochamad Ali Sobirin, Arata Fukushima, Yoshikuni Obata, Akimichi Saito, Naoki Ishimori, et al. The disruption of invariant natural killer T cells exacerbates cardiac hypertrophy and failure caused by pressure overload in mice. Experimental physiology. 2020. 105. 3. 489-501
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MISC (20件)：
  • Hideo Nambu, Shingo Takada, Satoshi Maekawa, Naoya Kakutani, Takaaki Furihata, Akimichi Saito, Arata Fukushima, Takashi Yokota, Shintaro Kinugawa. Empagliflozin Restores Lowered Exercise Endurance Capacity via the Activation of Skeletal Muscle Fatty Acid Oxidation in Murine Heart Failure. CIRCULATION. 2019. 140
  • Takaaki Furihata, Shintaro Kinugawa, Shingo Takada, Satoshi Maekawa, Takashi Katayama, Ryosuke Shirakawa, Hideo Nambu, Yoshikuni Obata, Katsuma Yamanashi, Naoya Kakutani, et al. The Outer Mitochondrial Membrane Protein, mitoNEET, Sustains Structure of Mitochondrial Cristae. CIRCULATION. 2018. 138
  • Hideo Nambu, Shingo Takada, Satoshi Maekawa, Junichi Matsumoto, Naoya Kakutani, Ryosuke Shirakawa, Takashi Katayama, Katsuma Yamanashi, Yoshikuni Obata, Takaaki Furihata, et al. Inhibition of Xanthine Oxidase in the Acute Phase Preserves Mitochondrial Function in Skeletal Muscle and Exercise Capacity in Heart Failure After Myocardial Infarction. CIRCULATION. 2018. 138
  • Ryosuke Shirakawa, Takashi Yokota, Takayuki Nakajima, Shingo Takada, Miwako Yamane, Akimichi Saito, Satoshi Maekawa, Naoya Kakutani, Takaaki Furihata, Junichi Matsumoto, et al. Excessive Mitochondrial Reactive Oxygen Species Emission From Circulating Blood Cells is Associated With Systemic Oxidative Stress, Severity of Heart Failure, and Exercise Intolerance. CIRCULATION. 2018. 138
  • Naoya Kakutani, Arata Fukushima, Takashi Yokota, Takashi Katayama, Ryosuke Shirakawa, Satoshi Maekawa, Hideo Nambu, Takahiro Abe, Takaaki Furihata, Junichi Matsumoto, et al. High Respiratory Exchange Ratio at Submaximal Exercise Level Predicts Adverse Clinical Outcomes in Patients With Heart Failure. CIRCULATION. 2017. 136
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特許 (1件)：
  • 樹状細胞を含有する医薬およびその製造方法
講演・口頭発表等 (6件)：
  • Alpha-galactosylceramide-pulsed human dendritic cells can ameliorate post-infarct cardiac remodeling and failure in mice
    (ESC Congress 2016 2016)
  • Circulating Invariant Natural Killer T Cells are Decreased in Patients With Chronic Heart Failure
    (CIRCULATION 2014)
  • Circulating Invariant Natural Killer T Cells are Decreased in Patients With Chronic Heart Failure
    (JOURNAL OF CARDIAC FAILURE 2014)
  • Activation of invariant natural killer T cells ameliorates the development of angiotensin II-mediated abdominal aortic aneurysm formation in obese ob/ob mice
    (ESC Congress 2014 2014)
  • Activation of Invariant Natural Killer T Cells Ameliorates the Development of Angiotensin II-Mediated Abdominal Aortic Aneurysm Formation in Obese ob/ob Mice
    (CIRCULATION 2013)
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