研究者
J-GLOBAL ID:202101009838282947   更新日: 2024年01月30日

倉島 公憲

Kurashima Kiminori
所属機関・部署:
職名: 特任助教
競争的資金等の研究課題 (2件):
  • 2019 - 2022 SWI/SNFクロマチン再構成異常を標的としたATR阻害剤によるがん治療基盤確立
  • 2015 - 2017 Y-family DNAポリメラーゼ阻害によるc-myc高発現がん治療基盤の確立
論文 (6件):
  • Kiminori Kurashima, Hideto Kashiwagi, Iwao Shimomura, Ayako Suzuki, Fumitaka Takeshita, Marianne Mazevet, Masahiko Harata, Takayuki Yamashita, Yusuke Yamamoto, Takashi Kohno, et al. SMARCA4 deficiency-associated heterochromatin induces intrinsic DNA replication stress and susceptibility to ATR inhibition in lung adenocarcinoma. NAR Cancer. 2020. 2. 2
  • Kiminori Kurashima. Polη, a Y-family translesion synthesis polymerase, promotes cellular tolerance of Myc-induced replication stress. Journal of cell science. 2018. 131. 12. jcs212183-jcs212183
  • Kiminori Kurashima. Acute HSF1 depletion induces cellular senescence through the MDM2-p53-p21 pathway in human diploid fibroblasts. Journal of cell science. 2018. 131. 9. jcs210724-jcs210724
  • Kiminori Kurashima. Both high-fidelity replicative and low-fidelity Y-family polymerases are involved in DNA rereplication. Molecular and cellular biology. 2014
  • Kiminori Kurashima. A uvs-5 strain is deficient for a mitofusin gene homologue, fzo1, involved in maintenance of long life span in Neurospora crassa. Eukaryotic cell. 2012. 12. 2. 233-243
もっと見る
MISC (1件):
  • Takayuki Sekimoto, Tsukasa Oda, Kiminori Kurashima, Fumio Hanaoka, Takayuki Yamashita. Y-family polymerases are involved in oncogene-induced aberrant replication. DNA REPAIR. 2015. 28. 144-144
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