Murakami A, Tsuji K, Isoda M, Matsuo M, Abe Y, Yasui M, Hitoshi Okamura, Tominaga K. Prolonged Light Exposure Induces Circadian Impairment in Aquaporin-4-Knockout Mice. Journal of biological rhythms. 2023. 38. 2. 208-214
Yingqi Wang, Ryosuke Suzuki, Atsushi Fujii, Kenji Ieki, Wakami Goda, Masato Yasui, Yoichiro Abe. A tyrosine-based YXXΦ motif regulates the degradation of aquaporin-4 via both lysosomal and proteasomal pathways and is functionally inhibited by a 10-amino-acid sequence within its C-terminus. The FEBS Journal. 2023
Akiko Imaizumi, Takayuki Obata, Jeff Kershaw, Yasuhiko Tachibana, Yoichiro Abe, Sayaka Shibata, Nobuhiro Nitta, Ichio Aoki, Masato Yasui, Tatsuya Higashi. Quantitative measurement of diffusion-weighted imaging signal using expression-controlled aquaporin-4 cells: Comparative study of 2-compartment and diffusion kurtosis imaging models. PLOS ONE. 2022. 17. 4
Yoichiro Abe, Masato Yasui. Aquaporin-4 in Neuromyelitis Optica Spectrum Disorders: A Target of Autoimmunity in the Central Nervous System. Biomolecules. 2022. 12. 4. 591-591
Kazuhisa Ishida, Kaoru Yamada, Risa Nishiyama, Tadafumi Hashimoto, Itaru Nishida, Yoichiro Abe, Masato Yasui, Takeshi Iwatsubo. Glymphatic system clears extracellular tau and protects from tau aggregation and neurodegeneration. The Journal of experimental medicine. 2022. 219. 3
Development of a new Neuromyelitis Optica model mice.
(The 18th World Congress of Basic and Clinical Pharmacology/The 91st Annual Meeting of the Japanese Pharmacological Society 2018)
Roles of the C-terminal domain in intracellular trafficking and degradation of AQP4.
(The 40th Annual Meeting of the Molecular Biology Society of Japan 2017)
Aquaporin-4 is not involved in chronic neuroinflammatory responses in 5xFAD mice.
(The 90th Annual Meeting of the Japanese Pharmacological Society 2017)
Glial responses to amyloid β accumulation in aquaporin-4-deficient Alzheimer’s disease model.
(The 39th Annual Meeting of the Japan Neuroscience Society 2016)
High avidity chimeric monoclonal antibodies against the extracellular domains of human aquaporin-4 competing with NMO-IgG.
(The 88th Annual Meeting of the Japanese Pharmacological Society 2015)
