Rchr

J-GLOBAL ID：200901043210029558 Update date: Apr. 17, 2024

Matsuyama Zenjiro

マツヤマゼンジロウ | Matsuyama Zenjiro

Affiliation and department：
Job title：助手

Research field (3)： Neurology , Pathobiochemistry , Neuroscience - general

Research keywords (2)：脳神経疾患の病態と治療 , Pathological mechanism & treatment of neurological disorders

Research theme for competitive and other funds (6)：

遺伝性神経疾患の遺伝子治療
遺伝性神経疾患の遺伝子診断法の開法
カルシウムチャネルの異常による神経疾患の病態メカニズムの解明
Gene therapy of hereditary neurological disorders
Gene analysis of hereditary neurological-disorders

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MISC (41)：

P/Q型電位依存性カルシウムチャネル異常による神経細胞死機構の検討。(共著). 第?回老年疾患研究会. 2000
Apoptosis induced by polyglutamine expansion of the P/Q type Ca²⁺ channel(共著). 30^th Annual Meeting Society for Neuroscience. 2000
Reduced voltage sensitivity of action of P/Q-type Ca²⁺ chomels is associated with the ataxic mouse mutation rolling Nagoya(tg rol). J. Neurosci. 2000. 20. 15. 5654-5662
Z Matsuyama, Y Izumi, M Kameyama, H Kawakami, S Nakamura. The effect of CAT trinucleotide interruptions on the age at onset of spinocerebellar ataxia type 1 (SCA1). JOURNAL OF MEDICAL GENETICS. 1999. 36. 7. 546-548
Y Kaseda, H Kawakami, Z Matsuyama, R Kumagai, M Toji, O Komure, M Nishimura, Y Izumi, F Udaka, M Kameyama, et al. Spinocerebellar ataxia type 6 in relation to CAG repeat length. ACTA NEUROLOGICA SCANDINAVICA. 1999. 99. 4. 209-212

ｍore...

Works (4)：

遺伝性神経疾患の遺伝子治療
2000 -
カルシウムチャネルの異常による神経疾患の病態メカニズムの解明
2000 -
Gene therapy of hereditary neurological disorders
2000 -
mechanism of neurological disorders due to the abnormalities of calcium channels.
2000 -

Professional career (1)：

Doctor(MD & PhD) (Hiroshima University)

Association Membership(s) (6)：

日本神経化学会 , 北米神経科学会 , 人類遺伝学会 , 日本内科学会 , 日本老年学会 , 日本神経学会

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