Rchr
J-GLOBAL ID:200901049838870292   Update date: Nov. 06, 2024

Takanori Matsui

マツイ タカノリ | Takanori Matsui
Affiliation and department:
Research field  (4): Food sciences ,  Chemical biology ,  Laboratory animal science ,  Cell biology
Research keywords  (9): physiologically functional food ,  Advanced Glycation End-products ,  Pigment Epithelium Derived Factor ,  peptide ,  aptamer ,  insulin resistance ,  diabetic complication ,  細胞生物学 ,  Cell Biology
Research theme for competitive and other funds  (15):
  • 2024 - 2027 Aptamer Raised Against Receptor for Advanced Glycation End Products (RAGE) Improves Survival Rate in Septic Acute Kidney Injury Mice
  • 2023 - 2026 Development of an Innovative Therapy Utilizing RAGE Aptamer to Achieve Zero Mortality in Neonatal Necrotizing Enterocolitis.
  • 2023 - 2026 Development of a novel therapeutic approach for diabetic nephropathy by nucleic acid medicine (DNA aptamer).
  • 2022 - 2025 内因性一酸化炭素COの産生調節機構の解明と新規メディカルガスによる治療基盤の構築
  • 2019 - 2022 Identification of structures of advanced glycation end-products responsible for proatherosclerotic effects - analysis using aptamers.
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Papers (243):
  • Michishige Terasaki, Hironori Yashima, Yusaku Mori, Tomomi Saito, Naoto Inoue, Takanori Matsui, Naoya Osaka, Tomoki Fujikawa, Makoto Ohara, Sho-Ichi Yamagishi. Glucose-Dependent Insulinotropic Polypeptide Inhibits AGE-Induced NADPH Oxidase-Derived Oxidative Stress Generation and Foam Cell Formation in Macrophages Partly via AMPK Activation. International journal of molecular sciences. 2024. 25. 17
  • Yusaku Mori, Michishige Terasaki, Naoya Osaka, Tomoki Fujikawa, Hironori Yashima, Tomomi Saito, Yurie Kataoka, Makoto Ohara, Yuichiro Higashimoto, Takanori Matsui, et al. DNA Aptamer Raised against Advanced Glycation End Products Improves Sperm Concentration, Motility, and Viability by Suppressing RAGE-Induced Oxidative Stress and Inflammation in Testes of Diabetic Mice. 2024
  • Yuta Nakamura, Ichiro Horie, Tadahiro Kitamura, Yoshiki Kusunoki, Kenro Nishida, Akane Yamamoto, Yushi Hirota, Tomoyasu Fukui, Yasutaka Maeda, Masae Minami, et al. Glucagon secretion and its association with glycaemic control and ketogenesis during sodium-glucose cotransporter 2 inhibition by ipragliflozin in people with type 1 diabetes: Results from the multicentre, open-label, prospective study. Diabetes, obesity & metabolism. 2024
  • Yosuke Ochi, Takanori Matsui, Keitaro Inoue, Kohei Monobe, Hiroshi Sakamoto, Shunsuke Aoki, Junichi Taira. Computational Screening and Experimental Validation of Inhibitor Targeting the Complex Formation of Grb14 and Insulin Receptor. Molecules. 2023. 29. 1. 298
  • 福井 智康, 山岸 昌一, 小原 信, 竹鼻 伸晃, 堀江 一郎, 中村 祐太, 阿比留 教生, 北村 忠弘, 楠 宜樹, 西田 健郎, et al. 1型糖尿病患者に対するSGLT-2阻害薬投与後のketogenesisとPEDFの関係. 日本先進糖尿病治療研究会雑誌. 2023. 17. 3. 154-154
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MISC (204):
  • 八島広典, 寺崎道重, 外川内亜美, 松井孝憲, 齋藤智美, 森雄作, 大坂直也, 小原信, 福井智康, 山岸昌一. Metformin suppresses the Cdk5-CD36 pathway via AMPK activation and antagonizes macrophage foam cell formation. 日本メイラード学会年会プログラム・抄録集. 2023. 33rd
  • 松井孝憲, 坂口達也, 東元祐一郎, 山岸昌一. スルフォラファンの抗AGEs-RAGE作用. 日本食品化学学会総会・学術大会講演要旨集. 2023. 29th
  • 松井孝憲, 東元祐一郎, 外川内亜美, 山岸昌一. DNA aptamers against AGE-RAGE axis inhibit growth and metastasis of malignant melanoma in mice. 日本分子生物学会年会プログラム・要旨集(Web). 2023. 46th
  • 古賀義法, 外川内亜美, 東元祐一郎, 西野友梨, 松井孝憲, 角間辰之, 秋葉純, 田中芳明, 八木実, 加治建, et al. 終末糖化産物受容体を阻害するRAGEアプタマーは敗血症マウスの生存率を改善する. 日本抗加齢医学会総会プログラム・抄録集. 2023. 23rd
  • 寺崎道重, 柴田佳太, 柴田佳太, 森雄作, 斎藤智美, 松井孝憲, 八島広典, 大坂直也, 藤川大輝, 高畑洋, et al. SMTP-44DはApoE欠損マウスの動脈硬化進展を抑制する. 日本抗加齢医学会総会プログラム・抄録集. 2023. 23rd
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Patents (3):
Education (4):
  • 2000 - 2004 Kyushu Institute of Technology
  • 1997 - 2000 Kyushu Institute of Technology
  • 1995 - 1997 Kyushu Institute of Technology School of Computer Science and Systems Engineering
  • 1990 - 1995 National Institute of Technology, Fukui College
Professional career (1):
  • (BLANK)
Association Membership(s) (7):
Japanese Society of Food Chemistry ,  日本抗加齢医学会 ,  JAPANESE SOCIETY OF NEPHROLOGY ,  THE MOLECULAR BIOLOGY SOCIETY OF JAPAN ,  NUCLEIC ACIDS THERAPEUTICS SOCIETY OF JAPAN ,  THE JAPAN DIABETES SOCIETY ,  The Japanese Biochemical Society
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