Rchr
J-GLOBAL ID:200901050195508548   Update date: Oct. 05, 2024

Katsuki Yoko

カツキ ヨウコ | Katsuki Yoko
Affiliation and department:
Research keywords  (5): ファンコニ貧血 ,  複製ストレス ,  ゲノム安定性 ,  ユビキチン化 ,  DNA修復
Research theme for competitive and other funds  (6):
  • 2020 - 2023 The role of SLFN11 gene that determines DNA damage sensitivity at the stalled replication forks
  • 2020 - 2023 BioIDシステムを利用したDNA修復因子SLX4の制御機構の解析
  • 2019 - 2020 BioIDシステムの構築によるICL修復因子ネットワークのプロテオミクス解析
  • 2017 - 2019 Elucidation of the ubiquitination pathway mediating recruitment of SLX4 during ICL repair
  • 2011 - 2016 Analysis of disorders defective in the mechanisms for fork stabilization
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Papers (16):
  • Anfeng Mu, Yusuke Okamoto, Yoko Katsuki, Minoru Takata. The role of SLFN11 in DNA replication stress response and its implications for the Fanconi anemia pathway. DNA Repair. 2024. 103733-103733
  • Erin Alvi, Ayako L. Mochizuki, Yoko Katsuki, Minori Ogawa, Fei Qi, Yusuke Okamoto, Minoru Takata, Anfeng Mu. Mouse Slfn8 and Slfn9 genes complement human cells lacking SLFN11 during the replication stress response. Communications Biology. 2023
  • Yoko Katsuki, Masako Abe, Seon Young Park, Wenwen Wu, Hiromasa Yabe, Miharu Yabe, Haico van Attikum, Shinichiro Nakada, Tomohiko Ohta, Michael M Seidman, et al. RNF168 E3 ligase participates in ubiquitin signaling and recruitment of SLX4 during DNA crosslink repair. Cell reports. 2021. 37. 4. 109879-109879
  • Yusuke Okamoto, Masako Abe, Anfeng Mu, Yasuko Tempaku, Colette B Rogers, Ayako L Mochizuki, Yoko Katsuki, Masato T Kanemaki, Akifumi Takaori-Kondo, Alexandra T Sobeck, et al. SLFN11 promotes stalled fork degradation that underlies the phenotype in Fanconi anemia cells. Blood. 2020. 137. 3. 336-348
  • Yoko Katsuki, Penny A. Jeggo, Yuki Uchihara, Minoru Takata, Atsushi Shibata. DNA double-strand break end resection: a critical relay point for determining the pathway of repair and signaling. Genome Instability & Disease. 2020. 1. 4. 155-171
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MISC (2):
Lectures and oral presentations  (6):
  • RNF168 E3 ligase mediates ubiquitin signaling and recruitment of SLX4 during replication-coupled DNA crosslink repair
    (2021)
  • Elucidation of the ubiquitination pathway mediating recruitment of SLX4 during ICL repair
    (The 62nd Annual Meeting of the Japanese Radiation Research Society 2019)
  • DNAクロスリンク修復因子SLX4のユビキチン化による制御機構.
    (日本放射線影響学会第61回大会 2018)
  • DNAクロスリンク切断酵素SLX4のユビキチン化による制御機構.
    (第41回日本分子生物学会年会 2018)
  • DNAクロスリンク修復因子SLX4のユビキチン化による制御機構
    (日本遺伝学会第90回大会 2018)
more...
Education (1):
  • 2004 - 2008 Tokyo Medical and Dental University Graduate School of Medical and Dental Sciences
Professional career (1):
  • 医学博士 (東京医科歯科大学)
Work history (8):
  • 2022/04 - 現在 Kyushu University
  • 2021/04 - 2022/03 京都大学大学院生命科学研究科附属放射線生物研究センター 特定講師
  • 2018/04 - 2021/03 Kyoto University Graduate School of Biostudies
  • 2018/02 - 2018/03 京都大学放射線生物研究センター 特定助教
  • 2014/04 - 2018/01 京都大学放射線生物研究センター 研究員
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Association Membership(s) (4):
日本薬学会 ,  日本癌学会 ,  THE JAPANESE RADIATION RESEARCH SOCIETY ,  THE MOLECULAR BIOLOGY SOCIETY OF JAPAN
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