Rchr
J-GLOBAL ID:200901087915260574   Update date: Jun. 08, 2023

SHIRATORI HIDETAKA

シラトリ ヒデタカ | SHIRATORI HIDETAKA
Affiliation and department:
Job title: Professor
Research field  (1): Developmental biology
Research theme for competitive and other funds  (1):
  • 2020 - 2023 左右非対称な内臓器官の形態変化のしくみとその役割
Papers (34):
  • CFAP53 regulates mammalian cilia-type motility patterns through differential localization and recruitment of axonemal dynein components. 2020
  • CFAP45 deficiency causes situs abnormalities and asthenospermia by disrupting an axonemal adenine nucleotide homeostasis module. Nature Communications. 2020
  • Role of Ca 2+ transients at the node of the mouse embryo in breaking of left-right symmetry. Science Advances. 2020
  • Loss of PYCR2 Causes Neurodegeneration by Increasing Cerebral Glycine Levels via SHMT2. Neuron. 2020
  • Nabeshima R, Nishimura O, Maeda T, Shimizu N, Ide T, Yashiro K, Sakai Y, Meno C, Kadota M, Shiratori H, et al. Loss of Fam60a, a Sin3a subunit, results in embryonic lethality and is associated with aberrant methylation at a subset of gene promoters. Elife. 2018. 7. pii: e36435
more...
Lectures and oral presentations  (1):
  • <I>Pycr2</I> KOマウスにおける脳神経系異常の解析
    (2019)
Professional career (1):
  • 医学 (大阪大学)
Association Membership(s) (2):
日本分子生物学会 ,  日本獣医学会
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