Rchr
J-GLOBAL ID:201801018720015409   Update date: Jul. 30, 2022

Yuto Shiode

Yuto Shiode
Affiliation and department:
Research field  (1): Gastroenterology
Papers (21):
  • Kazuhiro Murai, Takahiro Kodama, Hayato Hikita, Akiyoshi Shimoda, Makoto Fukuoka, Keisuke Fukutomi, Satoshi Shigeno, Yuto Shiode, Daisuke Motooka, Yuichiro Higuchi, et al. Inhibition of nonhomologous end joining-mediated DNA repair enhances anti-HBV CRISPR therapy. Hepatology communications. 2022
  • Yuto Shiode, Takahiro Kodama, Satoshi Shigeno, Kazuhiro Murai, Satoshi Tanaka, Justin Y. Newberg, Jumpei Kondo, Shogo Kobayashi, Ryoko Yamada, Hayato Hikita, et al. TNF receptor-related factor 3 inactivation promotes the development of intrahepatic cholangiocarcinoma through NF-κB-inducing kinase-mediated hepatocyte transdifferentiation. Hepatology. 2022
  • 塩出 悠登, 小玉 尚宏, 疋田 隼人, 阪森 亮太郎, 巽 智秀, 竹原 徹郎. Traf3の不活性化は肝細胞の分化転換を介した肝内胆管がん発がんを促進する. 日本癌学会総会記事. 2021. 80回. [E14-1]
  • Naoki Mizutani, Hayato Hikita, Yoshinobu Saito, Yuta Myojin, Katsuhiko Sato, Makiko Urabe, Tomohide Kurahashi, Yuto Shiode, Sadatsugu Sakane, Kazuhiro Murai, et al. Gab1 in livers with persistent hepatocyte apoptosis has an antiapoptotic effect and reduces chronic liver injury, fibrosis, and tumorigenesis. American journal of physiology. Gastrointestinal and liver physiology. 2021. 320. 6. G958-G968
  • 塩出 悠登, 小玉 尚宏, 竹原 徹郎. 肝癌の基礎研究と臨床応用 Traf3による分化転換を介した肝内胆管癌発症機構の解明. 肝臓. 2021. 62. Suppl.1. A190-A190
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MISC (22):
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Professional career (2):
  • M.D. (Osaka University)
  • Ph.D. (Osaka University)
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