TAKUYA SATO

Research field (1)： Anatomy

Papers (32)：

Mai Nakayachi, Junta Ito, Chiyomi Hayashida, Yoko Ohyama, Akemi Kakino, Mari Okayasu, Takuya Sato, Toru Ogasawara, Toshio Kaneda, Naoto Suda, et al. Lectin-like oxidized low-density lipoprotein receptor-1 abrogation causes resistance to inflammatory bone destruction in mice, despite promoting osteoclastogenesis in the steady state. BONE. 2015. 75. 170-182
Chiyomi Hayashida, Junta Ito, Mai Nakayachi, Mari Okayasu, Yoko Ohyama, Yoshiyuki Hakeda, Takuya Sato. Osteocytes Produce Interferon-beta as a Negative Regulator of Osteoclastogenesis. JOURNAL OF BIOLOGICAL CHEMISTRY. 2014. 289. 16. 11545-11555
Mari Okayasu, Mai Nakayachi, Chiyomi Hayashida, Junta Ito, Toshio Kaneda, Masaaki Masuhara, Naoto Suda, Takuya Sato, Yoshiyuki Hakeda. Low-density Lipoprotein Receptor Deficiency Causes Impaired Osteoclastogenesis and Increased Bone Mass in Mice because of Defect in Osteoclastic Cell-Cell Fusion. JOURNAL OF BIOLOGICAL CHEMISTRY. 2012. 287. 23. 19229-19241
Naoto Hada, Mari Okayasu, Junta Ito, Mai Nakayachi, Chiyomi Hayashida, Toshio Kaneda, Noritaka Uchida, Takamichi Muramatsu, Chihiro Koike, Masaaki Masuhara, et al. Receptor activator of NF-kappa B ligand-dependent expression of caveolin-1 in osteoclast precursors, and high dependency of osteoclastogenesis on exogenous lipoprotein. BONE. 2012. 50. 1. 226-236
Masaaki Masuhara, Takuya Sato, Naoto Hada, Yoshiyuki Hakeda. Protective protein/cathepsin A down-regulates osteoclastogenesis by associating with and degrading NF-kappa B p50/p65. JOURNAL OF BONE AND MINERAL METABOLISM. 2009. 27. 1. 46-56

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MISC (5)：

Lectures and oral presentations (41)：

スダチ果皮特有ポリメトキシフラボノイドsudachitinは破骨細胞形成およびLPS誘導炎症性骨破壊を抑制する
(日本骨代謝学会学術集会プログラム抄録集 2016)
レクチン様酸化LDL受容体-1(LOX-1)欠損は定常状態の破骨細胞形成を促進するが、LPS誘導炎症性骨破壊には抵抗性を示す
(Journal of Oral Biosciences Supplement 2015)
レクチン様酸化LDL受容体-1(LOX-1)欠損は定常状態における破骨細胞形成を促進するが、LPS誘導炎症性骨破壊に抵抗性をもたらす
(日本骨代謝学会学術集会プログラム抄録集 2015)
新たな初代骨細胞培養法を開発し、骨細胞がinterferon-βによって破骨細胞形成を抑制することを示す
(明海歯科医学 2013)
レクチン様酸化LDL受容体-1(LOX-1)の破骨細胞形成と炎症性骨破壊に対する役割の解明
(Journal of Oral Biosciences Supplement 2013)

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