Rchr
J-GLOBAL ID：202101009838282947
Update date: Jan. 30, 2024
Kurashima Kiminori
Kurashima Kiminori
Affiliation and department：
Research theme for competitive and other funds (2)：
- 2019 - 2022 Development of a therapeutic foundation for SWI/SNF chromatin remodeling complex defective cancer by ATR inhibitor
- 2015 - 2017 Development of a therapeutic foundation for c-Myc overexpressed cancer by depletion of Y-family DNA polymerase
Papers (6)：
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Kiminori Kurashima, Hideto Kashiwagi, Iwao Shimomura, Ayako Suzuki, Fumitaka Takeshita, Marianne Mazevet, Masahiko Harata, Takayuki Yamashita, Yusuke Yamamoto, Takashi Kohno, et al. SMARCA4 deficiency-associated heterochromatin induces intrinsic DNA replication stress and susceptibility to ATR inhibition in lung adenocarcinoma. NAR Cancer. 2020. 2. 2
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Kiminori Kurashima. Polη, a Y-family translesion synthesis polymerase, promotes cellular tolerance of Myc-induced replication stress. Journal of cell science. 2018. 131. 12. jcs212183-jcs212183
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Kiminori Kurashima. Acute HSF1 depletion induces cellular senescence through the MDM2-p53-p21 pathway in human diploid fibroblasts. Journal of cell science. 2018. 131. 9. jcs210724-jcs210724
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Kiminori Kurashima. Both high-fidelity replicative and low-fidelity Y-family polymerases are involved in DNA rereplication. Molecular and cellular biology. 2014
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Kiminori Kurashima. A uvs-5 strain is deficient for a mitofusin gene homologue, fzo1, involved in maintenance of long life span in Neurospora crassa. Eukaryotic cell. 2012. 12. 2. 233-243
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MISC (1)：
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Takayuki Sekimoto, Tsukasa Oda, Kiminori Kurashima, Fumio Hanaoka, Takayuki Yamashita. Y-family polymerases are involved in oncogene-induced aberrant replication. DNA REPAIR. 2015. 28. 144-144
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