Yamazaki Soh

ヤマザキソウ | Yamazaki Soh

Affiliation and department：
Job title： Associate Professor

Research field (5)： Pathobiochemistry , Medical biochemistry , Immunology , Molecular biology , Functional biochemistry

Research keywords (5)： IκBζ , 遺伝子発現調節 , 上皮細胞 , IL-17シグナリング , JunB

Research theme for competitive and other funds (18)：

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Papers (44)：

Soh Yamazaki. The nuclear NF-κB regulator IκBζ: updates on its molecular functions and pathophysiological roles. Cells. 2024. 13. 1467
Yuichi Tsuchiya, Takao Seki, Kenta Kobayashi, Sachiko Komazawa-Sakon, Shigeyuki Shichino, Takashi Nishina, Kyoko Fukuhara, Kenichi Ikejima, Hidenari Nagai, Yoshinori Igarashi, et al. Fibroblast growth factor 18 stimulates the proliferation of hepatic stellate cells, thereby inducing liver fibrosis. Nature communications. 2023. 14. 1. 6304-6304
Takashi Nishina, Yutaka Deguchi, Mika Kawauchi, Chen Xiyu, Soh Yamazaki, Tetuo Mikami, Hiroyasu Nakano. Interleukin 11 confers resistance to dextran sulfate sodium-induced colitis in mice. iScience. 2023. 26. 2. 105934-105934
Soh Yamazaki, Naohiro Inohara, Masaki Ohmuraya, Yousuke Tsuneoka, Hideo Yagita, Takaharu Katagiri, Takashi Nishina, Tetuo Mikami, Hiromasa Funato, Kimi Araki, et al. IκBζ controls IL-17-triggered gene expression program in intestinal epithelial cells that restricts colonization of SFB and prevents Th17-associated pathologies. Mucosal immunology. 2022. 15. 6. 1321-1337
Takashi Nishina, Yutaka Deguchi, Daisuke Ohshima, Wakami Takeda, Masato Ohtsuka, Shigeyuki Shichino, Satoshi Ueha, Soh Yamazaki, Mika Kawauchi, Eri Nakamura, et al. Interleukin-11-expressing fibroblasts have a unique gene signature correlated with poor prognosis of colorectal cancer. Nature communications. 2021. 12. 1. 2281-2281

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MISC (27)：

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Lectures and oral presentations (37)：

Fibroblast growth factor 18 stimulates the proliferation of hepatic stellate cells, thereby inducing liver fibrosis
(第52回免疫学会学術集会 2024)
Unraveling the Role of IL-11-Producing Fibroblasts in Colon Homeostasis
(The 46th Annual Meeting of the Molecular Biology Society of Japan 2023)
IκBζ controls IL-17-triggered gene expression program in intestinal epithelial cells that restricts colonization of SFB and prevents Th17-associated pathologies
(第77回東邦医学会総会 2023)
腸上皮細胞におけるIκBζの役割:IL-17応答性の遺伝子発現を介した腸内細菌叢の調節と自己免疫疾患の抑制
(第96回日本生化学会大会 2023)
細胞死不全マウスの免疫制御異常の解析
(令和5年度適応・修復領域若手主体の会議 2023)

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Education (2)：

Professional career (1)：

Work history (4)：

Awards (7)：

2023/11 - 令和5年度東邦医学会賞 IκBζ controls IL-17-triggered gene expression program in intestinal epithelial cells that restricts colonization of SFB and prevents Th17-associated pathologies
2022/12 - 第51回日本免疫学会学術集会ベストポスター賞 IκBζ regulates IL-17-triggered gene program in intestinal epithelial cells that restricts colonization of SFB and prevents autoimmune disorders
2022/03 - 令和3年度東邦大学創立60周年記念学術振興奨励金腸上皮細胞の調節を介した免疫応答の制御機構
2020/02 - 令和元年度額田奨学金
2016/09 - 2016年 (第24回) 日本生化学会JB論文賞 Glucocorticoid augments lipopolysaccharide-induced activation of the IκBζ-dependent genes encoding the anti-microbial glycoproteins lipocalin 2 and pentraxin 3

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Association Membership(s) (3)：

日本分子生物学会 , 日本生化学会 , THE JAPANESE SOCIETY FOR IMMUNOLOGY

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