Rchr
J-GLOBAL ID：200901037619056646
Update date: Aug. 27, 2020
Sakoda Hideyuki
サコダ ヒデユキ | Sakoda Hideyuki
Affiliation and department：
University of Miyazaki Faculty of Medicine, School of Medicine
About University of Miyazaki Faculty of Medicine, School of Medicine
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Detailed information
Research keywords (4)：
分子生物学
, 糖尿病
, molecular biology
, diabetes mellitus
Research theme for competitive and other funds (2)：
糖尿病学
diabetology
MISC (7)：
Activation of AMPK is essential for AICAR-induced glucose uptake by skeletal muscle but not a dipocytes. Am J Physiol Endocrinal Metab. 2002. 28(6):E1239-44
Activation of AMPK is essential for AICAR-induced glucose uptake by skeletal muscle but not a dipocytes. Am J Physiol Endocrinal Metab. 2002. 28(6):E1239-44
Dexamethasone-induced insulin resistance in 3T3-L1 a dipocytes is due to inhibition of glucose transport rather than insulin signal transduction. Diabetes. 2000. 49(10), 1700-8
Dexamethasone-induced insulin resistance in 3T3-L1 a dipocytes is due to inhibition of glucose transport rather than insulin signal transduction. Diabetes. 2000. 49(10), 1700-8
Hideyuki Sakoda, Takehide Ogihara, Motonobu Anai, Makoto Funaki, Kouichi Inukai, Hideki Katagiri, Yasushi Fukushima, Yukiko Onishi, Hiraku Ono, Yoshio Yazaki, et al. No correlation of plasma cell 1 overexpression with insulin resistance in diabetic rats and 3T3-L1 adipocytes. Diabetes. 1999. 48. 7. 1365-1371
ｍore...
Work history (1)：
Former Institution /Organization Endocrinology and Metabolism senior researcher
Association Membership(s) (2)：
日本内科学会
, 日本糖尿病学会
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