Rchr
J-GLOBAL ID：200901066683012342
Update date: Jun. 06, 2020
Yamamoto Ken'ichi
ヤマモト ケンイチ | Yamamoto Ken'ichi
Research field (2)：
Pathobiochemistry
, Cell biology
Research keywords (2)：
ストレス
, Radiation Biol. and Toxicology Cell Biology Molecular Biology
Research theme for competitive and other funds (2)：
2000 - ストレス応答における細胞内シグナル伝達機構
2000 - Cellular Signal transduction pathways involved in stress response
MISC (47)：
A Barzilai, KI Yamamoto. DNA damage responses to oxidative stress. DNA REPAIR. 2004. 3. 8-9. 1109-1115
A Barzilai, KI Yamamoto. DNA damage responses to oxidative stress. DNA REPAIR. 2004. 3. 8-9. 1109-1115
WS Wang, M Seki, M Otsuki, S Tada, N Takao, K Yamamoto, M Hayashi, M Honma, T Enomoto. The absence of a functional relationship between ATM and BLM, the components of BASC, in DT40 cells. BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR BASIS OF DISEASE. 2004. 1688. 2. 137-144
WS Wang, M Seki, M Otsuki, S Tada, N Takao, K Yamamoto, M Hayashi, M Honma, T Enomoto. The absence of a functional relationship between ATM and BLM, the components of BASC, in DT40 cells. BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR BASIS OF DISEASE. 2004. 1688. 2. 137-144
T Fukao, P Chen, J Ren, H Kaneko, GX Zhang, M Kondo, K Yamamoto, Y Furuichi, S Takeda, N Kondo, et al. Disruption of the BLM gene in ATM-null DT40 cells does not exacerbate either phenotype. ONCOGENE. 2004. 23. 8. 1498-1506
ｍore...
Education (4)：
- 1978 Kanazawa University
- 1978 Kanazawa University Graduate School, Division of Medicine
- 1972 Kanazawa University
- 1972 Kanazawa University Faculty of Medicine
Professional career (1)：
医学博士
Work history (1)：
Kanazawa University Cancer Research Institute, Cancer Research Institute
Awards (1)：
2001 - 第33回病態代謝研究会研究助成金
Association Membership(s) (3)：
日本癌学会
, 日本分子生物学会
, 日本免疫学会
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