Rchr
J-GLOBAL ID：200901069497992930   Update date: Aug. 19, 2024

Koide Hiroshi

コイデ ヒロシ | Koide Hiroshi
Affiliation and department：
Juntendo University

About Juntendo University


Research field  (3)： Developmental biology ,  Molecular biology ,  Functional biochemistry
Research keywords  (2)： がん ,  幹細胞
Research theme for competitive and other funds  (29)：
  • 2022 - 2025 Development of Neuroaxonal Regeneration Therapy Using Adipose Tissue-Derived Stem Cells in Anticancer Drug-Induced Peripheral Neuropathy
  • 2021 - 2024 Is Fat Regeneration with bFGF-added PRP True? -Building Evidence in Aesthetic Medicine
  • 2019 - 2022 Analysis of molecular basis of carcinogenesis by novel oncogene Zfp57
  • 2016 - 2018 新規がん遺伝子Zfp57による細胞がん化の分子機構の解析
  • 2015 - Zfp57/Nrf2経路による老化制御や幹細胞性維持の分子機構の解析
Show all
Papers (57)：
  • Emma Tabe Eko Niba, Hiroyuki Awano, Noriyuki Nishimura, Hiroshi Koide, Masafumi Matsuo, Masakazu Shinohara. Differential metabolic secretion between muscular dystrophy mouse-derived spindle cell sarcomas and rhabdomyosarcomas drives tumor type development. American Journal of Physiology-Cell Physiology. 2024. 327. 1. C34-C47
  • Chihiro Matsui, Hiroshi Koide, Tomomi Ikeda, Takako Ikegami, Takumi Yamamoto, Joseph M. Escandón, Arbab Mohammad, Tomoyuki Ito, Hiroshi Mizuno. Cytokines released from human adipose tissue-derived stem cells by bFGF stimulation: Effects of IL-8 and CXCL-1 on wound healing. Regenerative Therapy. 2024. 26. 401-406
  • Mizoue, Y, Ikeda, T, Ikegami, T, Riabets, O, Oishi, Y, Tobita,, M, Akutsu, H, Hattori, K, Heissig, B, Koide, H. The stem cell transcription factor ZFP296 transforms NIH3T3 cells and promotes anchorage-independent growth of cancer cells. INTERNATIONAL JOURNAL OF DEVELOPMENTAL BIOLOGY. 2023. 67. 4. 147-153
  • Hashimoto, R, Koide, H, Katoh, Y. Long-term glucocorticoid treatment increases CD204 expression by activating the MAPK pathway and enhances modified LDL uptake in murine macrophages. Journal of Pharmacological Sciences. 2023. 151. 1. 46-53
  • Hashimoto, R, Koide, H, Katoh, Y. MEK inhibitors increase the mortality rate in mice with LPS-induced inflammation through IL-12-NO signaling. Cell Death Discovery. 2023. 9. 1. 374-374
ｍore...
MISC (13)：
  • 米谷充弘, 出村昌史, 唐島成宙, 米田隆, 小出寛, 堀家慎一, 西本鉱嗣郎, 向井邦晃, 山岸正和, 武田仁勇. コルチゾール産生腫瘍の体細胞変異はDNAメチル化に影響しているか. 日本高血圧学会総会プログラム・抄録集. 2016. 39th. 331-331
  • 高村博之, 山口紫, 庄司泰弘, 田夛祐喜, 赤木紀之, 小出寛, 正司政寿, 中沼伸一, 林泰寛, 酒井清祥, et al. ES細胞特異的遺伝子Zfp57は癌遺伝子として機能する. 日本外科学会雑誌. 2014. 115. 臨増2. 927-927
  • 小出 寛, 孫 伝海, 浦 大樹, 高尾 幸成, 木下 圭太, 赤木 紀之, 中武 悠樹, 丹羽 仁史, 横田 崇. LIF/STAT3経路によるマウスES細胞の未分化性維持機構. 生化学. 2009. 81. 9. 837-837
  • LIF/STAT3 pathway. 2009. 64. 538-547
  • 中武 悠樹, 赤木 紀之, 小出 寛, 横田 崇, 丹羽 仁史. 未分化ES細胞のoct3/4遺伝子発現制御の分子学的基礎(MOLECULAR BASIS OF TIGHT REGULATION ON OCT3/4 GENE EXPRESSION IN UNDIFFERENTIATED ES CELLS). 日本発生生物学会・日本細胞生物学会合同大会要旨集. 2007. 40回・59回. 152-152
ｍore...
Lectures and oral presentations  (16)：
  • The effect of overexpression of regenerating islet-derived type4 isoform 2 on colon cancer cell proliferation.
    (125th ASCRS Annual Scientific Meeting 2024)
  • 苦味受容体の細胞膜での発現および細胞応答に関する解析
    (第95回日本生化学大会 2022)
  • ES特異的転写因子Zfp296の細胞がん化への関与
    (第95回日本生化学大会 2022)
  • ES細胞に発現している転写因子Zfp296は細胞のがん化を引き起こす
    (第94回日本生化学大会 2021)
  • Cytokines Released from Human Adipose-tissue-Derived Stem Cells By b-FGF Stimulation: Effects on Angiogenesis and Lymphatic Vessels Formation By IL-8 and CXCL1
    (Plastic and Reconstructive Surgery - Global Open 2021)
ｍore...
Education (4)：
  • - 1992 The University of Tokyo
  • - 1992 The University of Tokyo Graduate School, Division of Science
  • - 1987 The University of Tokyo
  • - 1987 The University of Tokyo
Professional career (1)：
  • 博士(理学) (東京大学)
Work history (2)：
  • 2015/04 - 現在 Juntendo University
  • 2001/09 - 2015/03 Kanazawa University Institute of Medical, Pharmaceutical and Health Sciences, Faculty of Medicine
Awards (3)：
  • 2011 - 北國がん基金
  • 2008 - 内藤記念特定研究助成金
  • 2003 - 本陣奨励金
Association Membership(s) (3)：
THE JAPANESE ASSOCIATION FOR MOLECULAR TARGET THERAPY OF CANCER ,  日本分子生物学会 ,  日本生化学会
※ Researcher’s information displayed in J-GLOBAL is based on the information registered in researchmap. For details, see here.

Return to Previous Page

TOP

BOTTOM