Rchr
J-GLOBAL ID：200901082999137231
Update date: Apr. 03, 2018
Saitoh Masao
サイトウ マサオ | Saitoh Masao
Affiliation and department：
Research field (2)：
Oral medicine (pathology)
, Oral medicine
Research theme for competitive and other funds (2)：
- TGF-BシグナルとIRES制御
- 顎口腔領域疾患におけるTGF-B因子の作用機構の解明
MISC (40)：
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Tobiume K., Saitoh M. and lchijo H. Activation of apoptosis signal-regulating kinase 1 by the stress-induced activating phosphorylation of pre-formed oligomer. J. Cell Physiol. 2002. 191, 95-l04
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Saitoh M., Pullen N., Brennan P., Cantrell D., Dennis P. B. and Thomas G. Regulation of an activated S6K1 variant reveals a novel mTOR phosphorylation site. J Biol Chem. 2002. 277, 20104-20112
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Anja Jaeschke, Masao Saitoh, Joerg Hardkamp, Wendy Roworth, Takahiro Nobukuni, Angela Hedges, Julian Sampson, George Thomas and Richard Lamb. TSC Tumor Suppressor Mediated S6 Kinase Inhibition by PI3K is mTOR Independent. J. Cell Biol. 2002
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Tobiume K., Saitoh M. and lchijo H. Activation of apoptosis signal-regulating kinase 1 by the stress-induced activating phosphorylation of pre-formed oligomer. J. Cell Physiol. 2002. 191, 95-l04
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Saitoh M., Pullen N., Brennan P., Cantrell D., Dennis P. B. and Thomas G. Regulation of an activated S6K1 variant reveals a novel mTOR phosphorylation site. J. Biol. Chem. J Biol Chem. 2002. 277, 20104-20112
ｍore...
Professional career (1)：
Association Membership(s) (4)：
歯科基礎医学会
, 日本分子生物学会
, 日本生化学会
, 日本癌学会
