Rchr
J-GLOBAL ID:200901087915260574
Update date: Nov. 19, 2024
SHIRATORI HIDETAKA
シラトリ ヒデタカ | SHIRATORI HIDETAKA
Affiliation and department:
Job title:
Professor
Research field (1):
Developmental biology
Research theme for competitive and other funds (1):
- 2020 - 2023 左右非対称な内臓器官の形態変化のしくみとその役割
Papers (34):
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CFAP53 regulates mammalian cilia-type motility patterns through differential localization and recruitment of axonemal dynein components. 2020
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CFAP45 deficiency causes situs abnormalities and asthenospermia by disrupting an axonemal adenine nucleotide homeostasis module. Nature Communications. 2020
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Role of Ca 2+ transients at the node of the mouse embryo in breaking of left-right symmetry. Science Advances. 2020
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Loss of PYCR2 Causes Neurodegeneration by Increasing Cerebral Glycine Levels via SHMT2. Neuron. 2020
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Nabeshima R, Nishimura O, Maeda T, Shimizu N, Ide T, Yashiro K, Sakai Y, Meno C, Kadota M, Shiratori H, et al. Loss of Fam60a, a Sin3a subunit, results in embryonic lethality and is associated with aberrant methylation at a subset of gene promoters. Elife. 2018. 7. pii: e36435
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Lectures and oral presentations (3):
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Shmt2 KOマウスにおける胚発生異常の解析
(第46回日本分子生物学会年会 2023)
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動物の器官が左右非対称に形成されるしくみ
(第166回日本獣医学会 2023)
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<I>Pycr2</I> KOマウスにおける脳神経系異常の解析
(第42回日本分子生物学会年会 2019)
Professional career (1):
Association Membership(s) (2):
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