Art
J-GLOBAL ID：200902222984041270
Reference number：05A0588126
Resistance to 9-β-D-Arabinofuranosyl-2-Fluoroadenine due to Reduced Incorporation into DNA from Competition by Excess Deoxyadenosine Triphosphate: Implications for Different Sensitivities to Nucleoside Analogues
過剰デオキシアデノシン三りん酸による競合からのDNA取り込み低下に因る9-β-D-アラビノフラノシル-2-フルオロアデニンへの耐性:ヌクレオシドアナログへの異なる感受性の意義
Author (4)：
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Material：
Volume：
81
Issue：
5
Page：
405-412
Publication year：
Jun. 15, 2005
JST Material Number：
F0888A
ISSN：
0925-5710
CODEN：
IJHEEY
Document type：
Article
Article type：
原著論文
Country of issue：
Germany, Federal Republic of (DEU)
Language：
ENGLISH (EN)
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JST classification (2)：
JST classification
Category name(code) classified by JST.
Nucleosides,nucleotides
, Basic research of antitumor(=antineoplastic)drugs
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Reference (27)：
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RAI, KR. Fludarabine compared with chlorambucil as primary therapy for chronic lymphocytic leukemia. N Engl J Med. 2000, 343, 1750-1757
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YANG, LY. Arabinosyl-2-fluoroadenine augments cisplatin cytotoxicity and inhibits cisplatin-DNA cross-link repair. Mol Pharmacol. 1995, 47, 1072-1079
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TAKAGI, K. Inhibition of repair of carboplatin-induced DNA damage by 9-β-D-arabinofuranosyl-2-fluoroadenine in quiescent human lymphocytes. Biochem Pharmacol. 2004, 68, 1757-1766
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YAMAUCHI, T. DNA repair initiated in chronic lymphocytic leukemia lymphocytes by 4-hydroperoxycyclophosphamide is inhibited by fludarabine and clofarabine. Clin Cancer Res. 2001, 7, 3580-3589
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PLUNKETT, W. Metabolism and action of fludarabine phosphate. Semin Oncol. 1990, 17, 3-17
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Terms in the title (9)：
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