A Synergistic Increase of Apoptosis Utilizing Fas Antigen Expression Induced by Low Doses of Anticancer Drug

AKIYAMA H; INO T; TOKUNAGA E; KATSUDA I; EZAKI K

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J-GLOBAL ID：200902235058979367 Reference number：03A0595277

A Synergistic Increase of Apoptosis Utilizing Fas Antigen Expression Induced by Low Doses of Anticancer Drug

抗癌剤の低用量で誘発したFas抗原発現を利用したアポトーシスの相乗的増加

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Material：
Volume： 51 Issue： 8 Page： 733-739 Publication year： Aug. 25, 2003
JST Material Number： Z0687A ISSN： 0047-1860 CODEN： RBYOAI Document type： Article
Article type：原著論文 Country of issue： Japan (JPN) Language： ENGLISH (EN)

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Basic research of antitumor(=antineoplastic)drugs

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Reference (14)：

DA SILVA, CP. Apoptosis as a mechanism of cell death induced by different chemotherapeutic drugs in human leukemic T-lymphocytes. Biochem Pharmacol. 1996, 51, 1331-1340
MICHEAU, O. Dimanche-Boitrel MT. Sensitization of cancer cells treated with cytotoxic drugs to fas-mediated cytotoxicity. J Natl Cancer Inst. 1997, 89, 783-789
FRIESEN, C. Involvement of the CD95 (APO-1/FAS) receptor/ligand system in drug-induced apoptosis in leukemia cells. Nat Med. 1996, 5, 574-577
MCGAHON, AJ. Chemotherapeutic drug-induced apoptosis in human leukaemic cells is independent of the Fas (APO-1/CD95) receptor/ligand system. Br J Haematol. 1998, 101, 539-547
NAKAMURA, S. Anti-Fas IgM monoclonal antibody enhances apoptosis induced by low-dose cytosine arabinoside. Anticancer Res. 1999, 19, 197-204

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