Art

J-GLOBAL ID：200902286305952535   Reference number：04A0131238

A new paradigm for the progression of advanced heart failure.

心不全の重症化機構-その新しい概念-

Author (3)： KAWADA TOMIE (新潟大 医歯学総合病院 薬剤部) ,  NAKAZAWA MIKIO (新潟大 医 保健学科) ,  TOYOOKA TERUHIKO (東大 医 器官病態内科)
Material： 日本薬理学雑誌  (日本薬理学会西南部会抄録)
Volume： 123  Issue： 2  Page： 55-62  Publication year： Feb. 01, 2004 
JST Material Number： G0740A  ISSN： 0015-5691  CODEN： NYKZA  Document type： Article
Article type： 文献レビュー  Country of issue： Japan (JPN)  Language： JAPANESE (JA)

Thesaurus term： *cardiac insufficiency ,...

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JST classification (1)： Basic medicine on cardiovascular system  (GJ01020K)

Reference (29)：

• 1) Toyo-oka T, Kawada T, Xi H, et al. Gene therapy prevents disruption of dystrophin-related proteins in a model of hereditary dilated cardiomyopathies in hamster. Heart Lung Circ. 2002;11:174-181.
• 2) Homburger F, Baker JR, Nixon CW, et al. Primary, generalized polymyopathy and cardiac necrosis in an inbred line of Syrian hamsters. Med Exp. 1962;6:339-345.
• 3) Sakamoto A, Ono K, Abe M, et al. Both hypertrophic and dilated cardiomyopathies are caused by mutation of the same gene, δ-sarcoglycan, in hamster: an animal model of disrupted dystrophin-associated glycoprotein complex. Proc Natl Acad Sci USA. 1997;94:13873-13878.
• 4) Hoffman EP, Brown RJ, Kunkel LM. Dystrophin: the protein product of the Duchenne muscular dystrophy locus. Cell. 1987;51:919-928.
• 5) Cox GF, Kunkel LM. Dystrophies and heart disease. Curr Opin Cardiol. 1997;12:329-343.

Terms in the title (3)： 心不全 ,  重症化 ,  概念