Rchr
J-GLOBAL ID:201401003563548956   Update date: Oct. 07, 2024

Asagiri Masataka

Asagiri Masataka
Affiliation and department:
Job title: 教授
Research field  (2): Experimental pathology ,  Orthopedics
Research theme for competitive and other funds  (15):
  • 2024 - 2028 破骨細胞の新たな調節メカニズム:炎症と細胞外マトリクスの相互作用の解明
  • 2024 - 2027 Interaction of PTH and Wnt/beta-catenin signaling on impaired fracture healing with aging
  • 2021 - 2024 PDZRN3による核内のパラスペックルを介した新たな心筋分化制御機構の解明
  • 2020 - 2024 炎症・死細胞因子と細胞外マトリクス連関を介した炎症性破骨細胞形成メカニズム
  • 2016 - 2019 Understanding the Formation and Activation Mechanism of Inflammatory Osteoclasts
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Papers (43):
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MISC (15):
  • Seiya Tanaka, Okamoto Asuka, Tsubaki Honoka, Seto Tetsuya, Sakai Hiroki, Honda Takeshi, Asagiri Masataka. Inhibitory effect of spermidine on differentiation of inflammatory osteoclasts. Proceedings for Annual Meeting of The Japanese Pharmacological Society. 2022. 95. 3-P-249
  • Hina Takegami, Nakamura Hiyori, Takeda Ayano, Matsuo Mayu, Sakai Hiroki, Honda Takeshi, Asagiri Masataka. Synthetic retinoid bexarotene inhibits differentiation of inflammatory osteoclasts. Proceedings for Annual Meeting of The Japanese Pharmacological Society. 2022. 95. 3-P-248
  • Ishida Chika, Suzuki Miren, Seto Tetsuya, Otsuka Marina, Sakai Hiroki, Takeiri Masatoshi, Tsunekage Yukino, Arai Keiko, Honda Takeshi, Kimura Yoshihide, et al. Suppression of M1 macrophage activation and osteoclast differentiation by Apocynaceae plant extract. Proceedings for Annual Meeting of The Japanese Pharmacological Society. 2022. 95. 3-P-164
  • 本田 健, 酒井 大樹, 乾 誠, 朝霧 成挙. 筋分化時の一過性DNA鎖切断に対する新たな制御機構 PDZRN3は筋芽細胞をアポトーシスから保護する. 山口医学. 2021. 70. 3. 135-136
  • 本田 健, 酒井 大樹, 乾 誠, 朝霧 成挙. 筋分化時の一過性DNA鎖切断に対する新たな制御機構 PDZRN3は筋芽細胞をアポトーシスから保護する. 山口医学. 2021. 70. 3. 135-136
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