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J-GLOBAL ID：201402295949200126   Reference number：14A1386974

Tissue Injury-induced Reactive Oxygen Species Cause Dysfunction of Parotid Acinar Cells via Erk-novel PKC Activation

組織障害誘導活性酸素種は,Erk-新規PKC活性化を介し耳下腺腺房細胞の機能障害を引起す

Author (3)： INOUE Daisuke (Nihon Univ. School of Dentistry at Matsudo, Chiba, JPN) ,  YOKOYAMA Megumi (Nihon Univ. School of Dentistry at Matsudo, Chiba, JPN) ,  KATSUMATA-KATO Osamu (Nihon Univ. School of Dentistry at Matsudo, Chiba, JPN)
Material： International Journal of Oral-Medical Sciences  (IJOMS)
Volume： 13  Issue： 1  Page： 6-11  Publication year： Jun. 2014 
JST Material Number： L4836A  ISSN： 1347-9733  Document type： Article
Article type： 原著論文  Country of issue： Japan (JPN)  Language： ENGLISH (EN)

Thesaurus term： *active oxygen ,...

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JST classification (2)： Basic dentistry(= odontology)  (GT01020O) ,  Basic research of enzyme preparations and enzyme inhibitor preparations  (GW21010W)

Substance index (4)： Ro-31-8220 ,  U-0126 ,  Goe-6983 ,  Diphenylene iodonium

Reference (19)：

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• 2.Baum BJ: Prospects for re-engineering salivary glands. Adv Dent Res, 14: 84-88, 2000.
• 3.Fujita-Yoshigaki J, Tagashira A, Yoshigaki T, Furuyama S, and Sugiya H: A primary culture of parotid acinar cells retaining capacity for agonists-induced amylase secretion and generation of new secretory granules. Cell Tissue Res, 320:455-464, 2005.
• 4.Fujita-Yoshigaki J, Matsuki-Fukushima M, and Sugiya H:Inhibition of Src and p38 MAP kinases suppresses the change of claudin expression induced on dedifferentiation of primary cultured parotid acinar cells. Am J Physiol Cell Physiol, 294:C774-C785, 2008.
• 5.Fujita-Yoshigaki J, Qi B, Narita T, and Sugiya H: Parotid acinar cells transiently change to duct-like cells during epithelial-mesenchymal transition. J Med Invest, 56 Suppl:258-259, 2009.

Terms in the title (10)： 組織 ,  障害 ,  誘導 ,  活性酸素種 ,  ERK ,  PKC ,  活性化 ,  耳下腺 ,  腺房細胞 ,  機能障害