CCAAT/enhancer binding protein α (C/EBPα)<sup>+</sup> M2 macrophages contribute to fibrosis in IgG4-related disease?

YAMAMOTO Motohisa; SHIMIZU Yui; TAKAHASHI Hiroki; YAJIMA Hidetaka; YOKOYAMA Yoshihiro; ISHIGAMI Keisuke; TABEYA Tetsuya; SUZUKI Chisako; MATSUI Mikiko; NAISHIRO Yasuyoshi; IMAI Kohzon; SHINOMURA Yasuhisa

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J-GLOBAL ID：201502211577224855 Reference number：15A0772522

CCAAT/enhancer binding protein α (C/EBPα)⁺ M2 macrophages contribute to fibrosis in IgG4-related disease?

CCAAT/エンハンサー結合蛋白質α(C/EBPα)⁺M2マクロファージはIgG4関連疾患において線維症に寄与するか?

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Volume： 25 Issue： 3 Page： 484-486 Publication year： May. 2015
JST Material Number： L5142A ISSN： 1439-7595 Document type： Article
Article type：短報 Country of issue： United Kingdom (GBR) Language： ENGLISH (EN)

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Immunologic diseases,allergic diseases in general.

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Friedman AD, Keefer JR, Kummalue T, Liu H, Wang QF, Cleaves R. Regulation of granulocyte and monocyte differentiation by CCAAT/enhancer binding protein a. Blood Cells Mol Dis. 2003; 31(3):338-41.
Xie H, Ye M, Feng R, Graf T. Stepwise reprogramming of B cells into macrophages. Cell. 2004;117(5):663-76.
Bussmann LH, Schubert A, Vu Math TP, De Andres L, Desbordes SC, Parra M, et al. A robust highly efficient immune cell reprogramming system. Cell Stem Cell. 2009;5(5):554-66.
Khosroshahi A, Carruthers MN, Deshpande V, Unizony S, Bloch DB, Stone JH. Rituximab for the treatment of IgG4-related disease: lessons from 10 consecutive patients. Medicine (Baltimore). 2012;91(1):57-66.

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CCAAT/enhancer binding protein α (C/EBPα)+ M2 macrophages contribute to fibrosis in IgG4-related disease?

CCAAT/enhancer binding protein α (C/EBPα)⁺ M2 macrophages contribute to fibrosis in IgG4-related disease?