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J-GLOBAL ID:201702260946515509   Reference number:17A0681552

Aldosterone excess may inhibit insulin secretion: A comparative study on glucose metabolism pre- and post-adrenalectomy in patients with primary aldosteronism

アルドステロン過剰はインスリン分泌を阻害する可能性がある:原発性アルドステロン症患者における副腎摘出術の前と後のグルコース代謝に関する比較研究
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Volume: 64  Issue:Page: 339-346(J-STAGE)  Publication year: 2017 
JST Material Number: F0625A  ISSN: 0918-8959  Document type: Article
Article type: 原著論文  Country of issue: Japan (JPN)  Language: ENGLISH (EN)
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Crinical medicine on endocrine system in general 
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Reference (26):
  • 1. Catena C, Colussi G, Nadalini E, Chiuch A, Baroselli S, et al. (2008) Cardiovascular outcomes in patients with primary aldosteronism after treatment. Arch Intern Med 168: 80-85.
  • 2. Milliez P, Girerd X, Plouin P-F, Blacher J, Safar ME, et al. (2005) Evidence for an increased rate of cardiovascular events in patients with primary aldosteronism. J Am Coll Cardiol 45: 1243-1248.
  • 3. Born-Frontsberg E, Reincke M, Rump LC, Hahner S, Diederich S, et al. (2009) Cardiovascular and cerebrovascular comorbidities of hypokalemic and normokalemic primary aldosteronism: Results of the German Conn’s registry. J Clin Endocrinol Metab 94: 1125-1130.
  • 4. American Diabetes Association (2014) Diagnosis and Classification of Diabetes Mellitus. Diabetes Care 37 Suppl 1: S81-90.
  • 5. Chen F, Liu J, Wang Y, Wu T, Shan W, et al. (2015) Aldosterone induces clonal β-cell failure through glucocorticoid receptor. Sci Rep 5: 13215.
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