Rchr
J-GLOBAL ID：201801000768129922
Update date: Oct. 31, 2024
Eguchi Tomoya
エグチ トモヤ | Eguchi Tomoya
Affiliation and department：
Research field (1)：
Molecular biology
Research theme for competitive and other funds (3)：
- 2022 - 2025 オルガネラ膜の損傷・断裂を普遍的に検知する膜貫通ドメイン
- 2019 - 2022 How do lysosomes sense stress and activate homeostasis mechanisms?
- 2016 - 2018 パーキンソン病病因遺伝子LRRK2のリソソーム恒常性維持における機能
Papers (9)：
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Yuto Nagashima, Tomoya Eguchi, Ikuko Koyama-Honda, Noboru Mizushima. Optogenetic tools for inducing organelle membrane rupture. 2024
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Tomoya Eguchi, Maria Sakurai, Yingxue Wang, Chieko Saito, Gen Yoshii, Thomas Wileman, Noboru Mizushima, Tomoki Kuwahara, Takeshi Iwatsubo. The V-ATPase-ATG16L1 axis recruits LRRK2 to facilitate lysosomal stress responses. Journal of Cell Biology. 2023. 223. 3. e202302067
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Hideaki Morishita, Tomoya Eguchi, Noboru Mizushima. A new insight into the lens: cytosolic PLAAT phospholipases degrade organelles to make the lens transparent. Autophagy. 2021. 17. 9. 2645-2647
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Tomoya Eguchi, Hideaki Morishita, Noboru Mizushima. Monitoring Autophagic Activity In Vitro and In Vivo Using the GFP-LC3-RFP-LC3ΔG Probe. Neuromethods. 2021. 171. 41-51
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Hideaki Morishita, Tomoya Eguchi, Satoshi Tsukamoto, Yuriko Sakamaki, Satoru Takahashi, Chieko Saito, Ikuko Koyama-Honda, Noboru Mizushima. Organelle degradation in the lens by PLAAT phospholipases. Nature. 2021. 592. 7855. 634-638
ｍore...
Books (1)：
Lectures and oral presentations (1)：
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Maintenance of lysosomal homeostasis by Parkinson-associated LRRK2 and Rab GTPases
(UK-Japan Neuroscience Symposium 2018)
Professional career (1)：
Work history (4)：
- 2018/04 - 現在 東京大学大学院 医学系研究科 分子生物学分野
- 2014/04 - 2018/03 東京大学大学院 医学系研究科 神経病理学分野
- 2008/04 - 2009/09 The University of Tokyo
- The University of Tokyo School of Medicine, Faculty of Medicine
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