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J-GLOBAL ID：201802241757603068   Reference number：18A0420536

Rasal3-mediated T cell survival is essential for inflammatory responses

Rasal3媒介T細胞生存は炎症反応に必須である【Powered by NICT】

Author (5)： Muro Ryunosuke (Department of Immunology and Pathology, Research Institute, National Center for Global Health and Medicine, Chiba 272-8516, Japan) ,  Nitta Takeshi (Department of Immunology and Pathology, Research Institute, National Center for Global Health and Medicine, Chiba 272-8516, Japan) ,  Kitajima Masayuki (Department of Immunology and Pathology, Research Institute, National Center for Global Health and Medicine, Chiba 272-8516, Japan) ,  Okada Toshiyuki (Department of Immunology and Pathology, Research Institute, National Center for Global Health and Medicine, Chiba 272-8516, Japan) ,  Suzuki Harumi (Department of Immunology and Pathology, Research Institute, National Center for Global Health and Medicine, Chiba 272-8516, Japan)
Material： Biochemical and Biophysical Research Communications  (BBRC)
Volume： 496  Issue： 1  Page： 25-30  Publication year： 2018 
JST Material Number： B0118A  ISSN： 0006-291X  Document type： Article
Article type： 原著論文  Country of issue： Netherlands (NLD)  Language： ENGLISH (EN)

Abstract/Point： Fine regulation of the Ras/mit...

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Thesaurus term： Th2 cell ,  *Ras protein ,  *T cell ,  MAP kinase ,  apoptosis ,  protein kinase ,  lymph node ,  protein ,  cell death ,  Th1 cell ,  T cell receptor
Semi thesaurus term： 活性化T細胞 ,  ナイーブT細胞 ,  MAPキナーゼカスケード ,  *炎症反応 , 【Automatic Indexing@JST】

Author keywords (3)： Rasal3 ,  Apoptosis ,  Contact hypersensitivity (CHS)

JST classification (2)： Cell physiology in general  (EF02010S) ,  Immunological reaction in general  (ED03010T)

Terms in the title (3)： T細胞 ,  生存 ,  炎症反応