Art

J-GLOBAL ID：201902223579046374   Reference number：19A1871742

糖尿病性神経障害の病態生物学-細胞から個体へ-

Author (2)： SANGO KAZUNORI (東京都医学総合研) ,  SUZUKI MARI (東京都医学総合研)
Material： 糖尿病合併症  (日本糖尿病合併症学会プログラム・抄録集)
Volume： 33  Issue： 2  Page： 237-240  Publication year： Jul. 25, 2019 
JST Material Number： L4623A  Document type： Article
Article type： 解説  Country of issue： Japan (JPN)  Language： JAPANESE (JA)

Thesaurus term： *diabetic neuropathy ,...
Semi thesaurus term： Drosophila ,...

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JST classification (2)： Basic neurology  (GN01020W) ,  Metabolic diseases,nutritional diseases in general.  (GD05010J)

Reference (5)：

• Ho EC, Lam KS, Chen YS, et al.: Aldose reductase-deficient mice are protected from delayed motor nerve conduction velocity, increased c-Jun NH2-terminal kinase activation, depletion of reduced glutathione, increased superoxide accumulation, and DNA damage. Diabetes 55: 1946~1953 (2006)
• Niimi N, Yako H, Takaku S, et al.: A spontaneously immortalized Schwann cell line from aldose reductase-deficient mice as a useful tool for studying polyol pathway and aldehyde metabolism. J Neurochem 144: 710~722 (2018)
• Himeno T, Kamiya H, Naruse K, et al.: Beneficial effects of exendin-4 on experimental polyneuropathy in diabetic mice. Diabetes 60: 2397~2406 (2011)
• Tsukamoto M, Niimi N, Sango K, et al.: Neurotrophic and neuroprotective properties of exendin-4 in adult rat dorsal root ganglion neurons: involvement of insulin and PI3 kinase/RhoA signaling. Histochem Cell Biol 144: 249~259 (2015)
• Nakamura S, Oba M, Suzuki M, et al.: Suppression of autophagic activity by Rubicon is a signature of aging. Nat Commun 10: 847 (2019)

Terms in the title (5)： 糖尿病性神経障害 ,  病態 ,  生物学 ,  細胞 ,  個体