Art

J-GLOBAL ID：202002227597642868   Reference number：20A2069792

Disruption of dystonin in Schwann cells results in late-onset neuropathy and sensory ataxia

シュワン細胞におけるジストニンの破壊は遅発性ニューロパチーおよび感覚性運動失調をもたらす【JST・京大機械翻訳】

Author (25)： Horie Masao (Division of Neurobiology and Anatomy, Graduate School of Medical and Dental Sciences, Niigata University, Niigata, Japan) ,  Horie Masao (Department of Morphological Sciences, Kagoshima University, Kagoshima, Japan) ,  Horie Masao (Department of Nursing, Niigata College of Nursing, Niigata, Japan) ,  Yoshioka Nozomu (Division of Neurobiology and Anatomy, Graduate School of Medical and Dental Sciences, Niigata University, Niigata, Japan) ,  Kusumi Satoshi (Department of Morphological Sciences, Kagoshima University, Kagoshima, Japan) ,  Sano Hiromi (Division of System Neurophysiology, National Institute for Physiological Sciences, Okazaki, Japan) ,  Sano Hiromi (Department of Physiological Sciences, SOKENDAI, Okazaki, Japan) ,  Kurose Masayuki (Division of Oral Physiology, Graduate School of Medical and Dental Sciences, Niigata University, Niigata, Japan) ,  Kurose Masayuki (Department of Physiology, School of Dentistry, Iwate Medical University, Morioka, Japan) ,  Watanabe-Iida Izumi (Department of Cellular Neurobiology, Brain Research Institute, Niigata University, Niigata, Japan) ,  Watanabe-Iida Izumi (Division of Oral Biochemistry, Graduate School of Medical and Dental Sciences, Niigata University, Niigata, Japan) ,  Hossain Ibrahim (Division of Neurobiology and Anatomy, Graduate School of Medical and Dental Sciences, Niigata University, Niigata, Japan) ,  Hossain Ibrahim (Department of Biochemistry and Molecular Biology, Jahangirnagar University, Savar, Dhaka, Bangladesh) ,  Chiken Satomi (Division of System Neurophysiology, National Institute for Physiological Sciences, Okazaki, Japan) ,  Chiken Satomi (Department of Physiological Sciences, SOKENDAI, Okazaki, Japan) ,  Abe Manabu (Department of Cellular Neurobiology, Brain Research Institute, Niigata University, Niigata, Japan) ,  Abe Manabu (Department of Animal Model Development, Brain Research Institute, Niigata University, Niigata, Japan) ,  Yamamura Kensuke (Division of Oral Physiology, Graduate School of Medical and Dental Sciences, Niigata University, Niigata, Japan) ,  Sakimura Kenji (Department of Cellular Neurobiology, Brain Research Institute, Niigata University, Niigata, Japan) ,  Sakimura Kenji (Department of Animal Model Development, Brain Research Institute, Niigata University, Niigata, Japan) ,  Nambu Atsushi (Division of System Neurophysiology, National Institute for Physiological Sciences, Okazaki, Japan) ,  Nambu Atsushi (Department of Physiological Sciences, SOKENDAI, Okazaki, Japan) ,  Shibata Masahiro (Department of Morphological Sciences, Kagoshima University, Kagoshima, Japan) ,  Takebayashi Hirohide (Division of Neurobiology and Anatomy, Graduate School of Medical and Dental Sciences, Niigata University, Niigata, Japan) ,  Takebayashi Hirohide (Center for Coordination of Research Facilities, Niigata University, Niigata, Japan)
Material： GLIA
Volume： 68  Issue： 11  Page： 2330-2344  Publication year： 2020 
JST Material Number： W0157A  ISSN： 0894-1491  Document type： Article
Article type： 原著論文  Country of issue： United States (USA)  Language： ENGLISH (EN)

Abstract/Point： Dystonin (Dst) is a causative ...

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Thesaurus term： mRNA ,  Heredity ,  gene ,  hereditary disease ,  axon ,  sensory nerve ,  Phenotype ,  myelin ,  macrophage ,  *Schwann cell ,  *Ataxia ,  *neuropathy ,  transgenic mouse ,  dystonia ,  Cre

Author keywords (5)： BPAG1/BP230 ,  glial cell ,  hereditary sensory and autonomic neuropathy Type 6 (HSAN-VI) ,  hypomyelination ,  macrophage infiltration

JST classification (2)： Basic neurology  (GN01020W) ,  Biological function  (EB03040U)

Terms in the title (6)： シュワン細胞 ,  ジストニン ,  破壊 ,  遅発性ニューロパチー ,  感覚 ,  運動失調