土屋貴大

ツチヤアツヒロ | Tsutiya Atsuhiro

所属機関・部署：
職名：助教

研究分野 (3件)：分子生物学 , 動物生理化学、生理学、行動学 , 神経科学一般

研究キーワード (8件)：質量分析 , シナプス , タンパク質間相互作用 , 近位依存性ビオチン標識 , 生化学 , 免疫組織化学 , 細胞培養 , 神経科学

競争的資金等の研究課題 (2件)：

論文 (16件)：

Suzuka Shiono, Atsuhiro Tsutiya, Ritsuko Ohtani-Kaneko. Early Detection of Male-Predominant Phenotypes in the Pattern of Ultrasonic Vocalizations Emitted by Autism Spectrum Disorder Model (Crmp4-Knockout) Mice. Brain sciences. 2022. 12. 5
Atsuhiro Tsutiya, Mitsumi Arito, Takuma Tagashira, Masaaki Sato, Kazuki Omoteyama, Toshiyuki Sato, Naoya Suematsu, Manae S. Kurokawa, Tomohiro Kato. Layilin promotes mitochondrial fission by cyclin-dependent kinase 1 and dynamin-related protein 1 activation in HEK293T cells. Biochemical and Biophysical Research Communications. 2021. 549. 143-149
Omoteyama K, Sato T, Sato M, Tsutiya A, Arito M, Suematsu N, Kurokawa MS, Kato T. Identification of novel substrates of a disintegrin and metalloprotease 17 by specific labeling of surface proteins. Heliyon. 2020. 6. 12. e05804
Ushimaru S, Arito M, Tsutiya A, Sato T, Omoteyama K, Sato M, Suematsu N, Kurokawa SM, Kamijo-Ikemori A, Shibagaki Y, et al. Roles of Layilin in Regulation of Low-Density Lipoprotein Receptor in Malignant Glioma Cells. Journal of St. Marianna University. 2020
Kaji T, Arito M, Tsutiya A, Sase T, Onodera H, Sato T, Omoteyama K, Sato M, Suematsu N, Kurokawa MS, et al. Layilin enhances the invasive ability of malignant glioma cells via SNAI1 signaling. Brain Res. 2019. 1719. 140-147

MISC (13件)：

講演・口頭発表等 (20件)：

Substrate-specific neurite growth controlled by Ndr2 kinase through α1 integrin expression
(11th FENS Forum of Neuroscience, Berlin 2018 2018)
The functional alternations caused by Crmp4 mutation and their similarities to ASD characteristics and sexual dimorphism
(Collaboration Seminar on Development, Physiology & Ecology At Hakusan Campus, Toyo University 2018)
The function of CRMP4 in the brain development -Mainly focused on the olfactory bulb-
(IUPUI International Scientific Exchange Symposium 2015)
Collapsin response mediator protein 4 (CRMP4) knockout mice showed physiological alterations related to olfactory function
(Society for Neuroscience 2015 2015)
Deletion of Crmp4 results in altered morphology and physiology in the olfactory bulb.
(第120回日本解剖学会総会全国学術集会・第92回日本生理学会大会合同大会 2015)

学歴 (3件)：

学位 (1件)：

経歴 (5件)：

受賞 (1件)：

所属学会 (5件)：

日本動物学会 , 日本解剖学会 , 日本味と匂学会 , 北米神経科学会 , 日本神経科学学会

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土屋 貴大