Rchr
J-GLOBAL ID:200901090554704147
Update date: Jan. 17, 2024
Shi Kun
シークン | Shi Kun
Affiliation and department:
旧所属 滋賀医科大学 医学部附属病院
About 旧所属 滋賀医科大学 医学部附属病院
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Job title:
大学院学生(博士課程)
Research field (1):
Metabolism and endocrinology
Research keywords (4):
Endocrinology
, Diabetes Metabolism
, 内科・内分泌学
, 糖尿病学代謝学
Research theme for competitive and other funds (2):
2004 - 2006 肝臓におけるPTP1Bによるインスリン抵抗性の分子機構の解明
Molecular mechanism of insulin resistance in overexpression of PTPIB in liver
MISC (13):
Shi Kun, Egawa Katsuya, Maegawa Hiroshi, Nakamura Takaaki, Ugi Satoshi, Nishio Yoshihiko, Kashiwagi Atsunori. Protein-Tyrosine Phosphatase 1 B Associates with Insulin Receptor and Negatively Regulates Insulin Signaling without Receptor Internalization. J Biochem (Tokyo). 2004. 136(1):89-96. 1. 89-96
Protein phosphatase 2A negatively regulates insulin's metabolic signaling pathway by inhibiting Akt (protein kinase B) activity in 3T3-L1 adipocytes. Mol Cell Biol. 2004. Oct;24(19):8778-89
Protein-tyrosine phosphatase 1B associates with insulin receptor and negatively regulates insulin signaling without receptor internalization. J Biochem (Tokyo). 2004. Jul;136(1):89
Protein phosphatase 2A negatively regulates insulin's metabolic signaling pathway by inhibiting Akt (protein kinase B) activity in 3T3-L1 adipocytes. Mol Cell Biol. 2004. Oct;24(19):8778
S Shimizu, S Ugi, H Maegawa, K Egawa, Y Nishio, T Yoshizaki, K Shi, Y Nagai, K Morino, K Nemoto, et al. Protein-tyrosine phosphatase 1B as new activator for hepatic lipogenesis via sterol regulatory element-binding protein-1 gene expression. JOURNAL OF BIOLOGICAL CHEMISTRY. 2003. 278. 44. 43095-43101
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Education (3):
- 1998 中国哈尓(]G2332[)(ハルビン)医科大学 医学研究科 産婦人科学
- 1998 Harbin Medical University Graduate School, Division of Medical Science Obsterics and Gynecology
- 1995 中国ハルビン医科大学 医学系研究科 臨床医学
Professional career (1):
master
Work history (2):
1998 - 1999 客員研究員
1998 - 1999 Foreign researcher
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