Loss of imprinting of a paternally expressed transcript, with antisense orientation to K<sub>V</sub>LQT1, occurs frequently in Beckwith-Wiedemann syndrome and is independent of insulin-like growth factor II imprinting.

LEE M P; DEBAUN M R; MITSUYA K; GALONEK H L; BRANDENBURG S; OSHIMURA M; FEINBERG A P

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J-GLOBAL ID：200902151401451048 Reference number：99A0644854

Loss of imprinting of a paternally expressed transcript, with antisense orientation to K_VLQT1, occurs frequently in Beckwith-Wiedemann syndrome and is independent of insulin-like growth factor II imprinting.

KvLQT1へのアンチセンス開始を伴う父型発現転写物のインプリンティング消失はBeckwith-Wiedemann症候群で頻発し,IGF2インプリンティングとは無関係である

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Volume： 96 Issue： 9 Page： 5203-5208 Publication year： Apr. 27, 1999
JST Material Number： D0387A ISSN： 0027-8424 Document type： Article
Article type：原著論文 Country of issue： United States (USA) Language： ENGLISH (EN)

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Congenital diseases,deformities in general.

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Loss of imprinting of a paternally expressed transcript, with antisense orientation to KVLQT1, occurs frequently in Beckwith-Wiedemann syndrome and is independent of insulin-like growth factor II imprinting.

Loss of imprinting of a paternally expressed transcript, with antisense orientation to K_VLQT1, occurs frequently in Beckwith-Wiedemann syndrome and is independent of insulin-like growth factor II imprinting.