Characteristics of Apoptosis in HCT116 Colon Cancer Cells Induced by Deoxycholic Acid

YUI Satoko; SAEKI Tohru; KANAMOTO Ryuhei; IWAMI Kimikazu

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J-GLOBAL ID：200902218590999250 Reference number：05A0742368

Characteristics of Apoptosis in HCT116 Colon Cancer Cells Induced by Deoxycholic Acid

デオキシコール酸により誘導されたHCT116結腸癌細胞のアポトーシスの特性

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Material：
Volume： 138 Issue： 2 Page： 151-157 Publication year： Aug. 01, 2005
JST Material Number： F0286A ISSN： 0021-924X CODEN： JOBIAO Document type： Article
Article type：原著論文 Country of issue： Japan (JPN) Language： ENGLISH (EN)

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Liver , Chemistry,biochemistry and pathology of tumors(=neoplasms) , Cell physiology in general

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Reference (28)：

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HIGUCHI, H. Bile acid regulation of hepatic physiology : IV. Bile acids and death receptors. Amer. J. Physiol. Gastrointest. Liver Physiol. 2003, 284, G734-G738
SCHLOTTMAN, K. Characterization of bile salt-induced apoptosis in colon cancer cell lines. Cancer Res. 2000, 60, 4270-4276
QIAO, L. Deoxycholic acid (DCA) causes ligand-independent activation of epidermal growth factor receptor (EGFR) and FAS receptor in primary hepatocytes : inhibition of EGFR/mitogen-activated protein kinase-signaling module enhances DCA-induces apoptosis. Mol. Biol. Cell. 2001, 12, 2629-2645
GRAF, D. Taurolithocholic acid-3 sulfate induces CD95 trafficking and apoptosis in a c-Jun N-terminal kinase-dependent manner. Gastroenterology. 2002, 122, 1411-1427

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