ID1 Is Down-Regulated by Hepatocyte Growth Factor via ERK-Dependent and ERK-Independent Signaling Pathways, Leading to Increased Expression of p16<sup>INK4a</sup> in Hepatoma Cells

USHIO Kazutaka; HASHIMOTO Tomio; KITAMURA Naomi; TANAKA Toshiaki

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J-GLOBAL ID：200902257419270329 Reference number：09A1228813

ID1 Is Down-Regulated by Hepatocyte Growth Factor via ERK-Dependent and ERK-Independent Signaling Pathways, Leading to Increased Expression of p16^INK4a in Hepatoma Cells

Id1はERK依存的およびERK-独立的シグナル経路を通して肝細胞成長因子によって下方制御され肝細胞腫細胞においてp16^INK4aの過剰発現に至る

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Volume： 7 Issue： 7 Page： 1179-1188 Publication year： Jul. 2009
JST Material Number： W0166B ISSN： 1541-7786 Document type： Article
Country of issue： United States (USA) Language： ENGLISH (EN)

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ID1 Is Down-Regulated by Hepatocyte Growth Factor via ERK-Dependent and ERK-Independent Signaling Pathways, Leading to Increased Expression of p16INK4a in Hepatoma Cells

ID1 Is Down-Regulated by Hepatocyte Growth Factor via ERK-Dependent and ERK-Independent Signaling Pathways, Leading to Increased Expression of p16^INK4a in Hepatoma Cells