J-GLOBAL ID:201001085951643577   Update date: Dec. 02, 2019


Affiliation and department:
Job title: Professor
Homepage URL  (2): http://news7a1.atm.iwate-u.ac.jp/cgi-bin/list/list.cgi?id=134http://news7a1.atm.iwate-u.ac.jp/cgi-bin/list/list.cgi?id=134
Research field  (4): Laboratory animal science ,  Genetics/Genome dynamics ,  Developmental biology ,  Basic veterinary science/Basic zootechnical science
Research keywords  (3): Laboratory animal science ,  Model animals ,  Bone and cartilage metabolism
Research theme for competitive and other funds  (2):
  • Understanding the regulatory mechanism of osteoblast and chondrocyte differentiation with the use of animal models
  • Understanding the pathologic mechanism of bone and joint diseases with the use of animal models
Papers (44):
  • Furuichi T, Tsukamoto M, Saito M, Sato Y, Oiji N, Yagami K, Fukumura R, Gondo Y, Guo L, Ikegawa S, Yamamori Y, Tomii K. Crim1^C140S mutant mice reveal the importance of cysteine 140 in the internal region 1 of CRIM1 for its physiological functions. Mamma Genome. 2019. 30. 329-338
  • Sasaki S, Tsukamoto M, Saito M, HojyoS, Fukada T, Takami M and Furuichi T. Disruption of the mouse Slc39a14 gene encoding zinc transporter ZIP14 is associated with decreased bone mass, likely caused by enhanced bone resorption. FEBS Open Bio. 2018. 8. 655-663
  • Ichimura S, Sasaki S, Murata T, Fukumura R, Gondo Y, Ikegawa S, Furuichi T. An ENU-induced p.C225S missense mutation in the mouse Tgfb1 gene does not cause Camurati-Engelmann disease-like skeletal phenotypes. 2017. 66. 137-144
  • Moriishi T, Fukuyama R, Miyazaki T, Furuichi T, Ito M and Komori T. Overexpression of BCLXL in osteoblasts inhibits osteoblast apoptosis and increases bone volume and strength with a normal bone structure. J Bone Miner Res. 2016. 31. 7. 1366-1380
  • Kimura M, Ichimura S, Sasaki K, Masuya H, Suzuki T, Wakana S, Ikegawa S and Furuichi T. Endoplasmic reticulum stress-mediated apoptosis contributes to a skeletal dysplasia resembling platyspondylic lethal skeletal dysplasia, Torrance type, in a novel Col2a1 mutant mouse line. Biochem Biophys Res Commun. 2015. 468. 86-91
MISC (1):
  • TATSUYA FURUICHI. Genetic skeletal disorders with collagen abnormalities and endoplasmic reticulum stress. THE BONE. 2018. 32. 2. 209-214
Lectures and oral presentations  (16):
  • トランスジェニックマウス作製による転写因子SP7の骨芽細胞分化における機能解析
    (第154回日本獣医学会 2012)
  • Gdf5変異マウスにおける変形性関節症の発症機序の検討.
    (第59回日本実験動物学会総会 2012)
  • 亜鉛トランスポーター欠損マウスを用いた亜鉛による骨量制御機構の研究
    (第156回日本獣医学会 2013)
  • 亜鉛トランスポーター欠損マウスを用いた亜鉛による骨量制御機構の解明
    (第60回日本実験動物学会 2013)
  • TGFB1遺伝子のLAPドメインコード領域に変異を持つマウスの同定.
    (第157回日本獣医学会 2014)
Education (1):
  • - 1992 Hokkaido University Faculty of Veterinary Medicine
Professional career (1):
  • Ph.D (Osaka University)
Work history (5):
  • 1992 - 2000 Chugai Pharmaceutical Co. Ltd
  • 2000 - 2004 Osaka University School of Medicine
  • 2004 - 2010 RIKEN
  • 2010 - 2012 Jikei University School of Medicine
  • 2012 - 現在 Iwate University
Association Membership(s) (6):
JAPAN VETERINARY MEDICAL ASSOCIATION ,  Japanese Association for Laboratory Animal Science ,  Japanese Association for Laboratory Animal Medicine ,  The Japanese Society of Veterinary Science ,  The Japanese Society for Bone and Mineral Research ,  The Japan Society of Human Genetics
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