Art

J-GLOBAL ID：201002266331144005   Reference number：10A0397939

Overexpression of Sall1 in vivo leads to reduced body weight without affecting kidney development

in vivoにおけるSall1の過剰発現は腎臓発生に影響することなく体重低減をもたらす

Author (4)： JIANG Qing (Kumamoto Univ., Kumamoto, JPN) ,  FUJIMURA Sayoko (Kumamoto Univ., Kumamoto, JPN) ,  KOBAYASHI Chiyoko (Kumamoto Univ., Kumamoto, JPN) ,  NISHINAKAMURA Ryuichi (Kumamoto Univ., Kumamoto, JPN)
Material： Journal of Biochemistry  (JB)
Volume： 147  Issue： 3  Page： 445-450  Publication year： Mar. 01, 2010 
JST Material Number： W1920A  ISSN： 0021-924X  CODEN： JOBIAO  Document type： Article
Article type： 原著論文  Country of issue： United Kingdom (GBR)  Language： ENGLISH (EN)

Thesaurus term： body weight ,...
Semi thesaurus term： Cre recombinase ,...

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JST classification (3)： Gene expression  (EC02040Q) ,  Development(=generation)and differentiation  (EJ16020W) ,  Congenital diseases,deformities in general.  (GD03010V)

Reference (21)：

• KUHNLEIN, R. P. spalt encodes an evolutionarily conserved zinc finger protein of novel structure which provides homeotic gene function in the head and tail region of the Drosophila embryo. EMBO J. 1994, 13, 168-179
• NISHINAKAMURA, R. Murine homolog of Sall1 is essential for ureteric bud invasion in kidney development. Development. 2001, 128, 3105-3115
• NISHINAKAMURA, R. Stem cells in the embryonic kidney. Kidney Int. 2008, 73, 913-917
• NISHINAKAMURA, R. Essential roles of Sall family genes in kidney development. J. Physiol. Sci. 2006, 56, 131-136
• SAKAKI-YUMOTO, M. The murine homolog of SALL4, a causative gene in Okihiro syndrome, is essential for embryonic stem cell proliferation, and cooperates with Sall1 in anorectal, heart, brain and kidney development. Development. 2006, 133, 3005-3013

Terms in the title (3)： 過剰発現 ,  腎臓発生 ,  体重