Art

J-GLOBAL ID：201102243212992607   Reference number：11A1265910

Glucose-incretin interaction revisited [Review]

グルコース-インクレチン相互作用再考

Author (5)： ISHII Hiroaki (Dep. of Diabetes, Endocrinology and Metabolism, Shinshu Univ. School of Medicine, Matsumoto 390-8621, JPN) ,  SATO Yoshihiko (Dep. of Diabetes, Endocrinology and Metabolism, Shinshu Univ. School of Medicine, Matsumoto 390-8621, JPN) ,  TAKEI Masahiro (Dep. of Diabetes, Endocrinology and Metabolism, Shinshu Univ. School of Medicine, Matsumoto 390-8621, JPN) ,  NISHIO Shinichi (Dep. of Diabetes, Endocrinology and Metabolism, Shinshu Univ. School of Medicine, Matsumoto 390-8621, JPN) ,  KOMATSU Mitsuhisa (Dep. of Diabetes, Endocrinology and Metabolism, Shinshu Univ. School of Medicine, Matsumoto 390-8621, JPN)
Material： Endocrine Journal  (Endocr J)
Volume： 58  Issue： 7  Page： 519-525 (J-STAGE)  Publication year： 2011 
JST Material Number： F0625A  ISSN： 0918-8959  Document type： Article
Article type： 文献レビュー  Country of issue： Japan (JPN)  Language： ENGLISH (EN)

Thesaurus term： *peptide hormone ,...
Semi thesaurus term： cellular signal transduction ,...

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JST classification (1)： Pancreas  (EJ06050Y)

Substance index (2)： cAMP ,  Glucose

Reference (41)：

• SAAD, MF. A two-step model for development of non-insulin-dependent diabetes. Am J Med. 1991, 90, 2, 229-235
• KADOWAKI, T. Risk factors for worsening to diabetes in subjects with impaired glucose tolerance. Diabetologia. 1984, 26, 1, 44-49
• WEYER, C. The natural history of insulin secretory dysfunction and insulin resistance in the pathogenesis of type 2 diabetes mellitus. J Clin Invest. 1999, 104, 6, 787-794
• TANIGUCHI, A. Pathogenic factors responsible for glucose intolerance in patients with NIDDM. Diabetes. 1992, 41, 12, 1540-1546
• SATO, Y. Diminution of early insulin response to glucose in subjects with normal but minimally elevated fasting plasma glucose. Evidence for early beta-cell dysfunction. Diabet Med. 2002, 19, 7, 566-571

Terms in the title (3)： グルコース ,  インクレチン ,  相互作用