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J-GLOBAL ID:201502220706292683   Reference number:15A0828258

Suppressing effect of cordycepin on the lipopolysaccharide-induced nitric oxide production in RAW 264.7 cells

RAW264.7細胞におけるリポ多糖誘導の一酸化窒素生産でのコルジセピンの抑制効果
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Volume: 79  Issue:Page: 1021-1025  Publication year: Jun. 2015 
JST Material Number: G0021A  ISSN: 0916-8451  CODEN: BBBIEJ  Document type: Article
Article type: 短報  Country of issue: United Kingdom (GBR)  Language: ENGLISH (EN)
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Pharmacology of antibiotics in general 
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Reference (28):
  • Palmer RM, Ferrige AG, Moncada S. Nitric oxide release accounts for the biological activity of endothelium-derived relaxing factor. Nature. 1987;327:524-526.
  • Nathan C. Nitric oxide as a secretory product of mammalian cells. FASEB J. 1992;6:3051-3064.
  • Lind L. Circulating markers of inflammation and atherosclerosis. Atherosclerosis. 2003;169:203-214.
  • Baumann H, Gauldie J. Regulation of hepatic acute phase plasma protein genes by hepatocyte stimulating factors and other mediators of inflammation. Mol. Biol. Med. 1990;7:147-159.
  • Kwak HJ, Song JS, Heo JY, Yang SD, Nam JY, Cheon HG. Roflumilast inhibits lipopolysaccharide-induced inflammatory mediators via suppression of nuclear factor-kappaB, p38 mitogen-activated protein kinase, and c-Jun NH2-terminal kinase activation. J. Pharmacol. Exp. Ther. 2005;315:1188-1195.
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