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J-GLOBAL ID：201902268990665325   Reference number：19A1970393

CBP/p300 antagonises EGFR-Ras-Erk signalling and suppresses increased Ras-Erk signalling-induced tumour formation in mice

CBP/p300はEGFR-Ras-ERKシグナル伝達を拮抗し,マウスにおけるRas-ERKシグナル伝達誘導腫瘍形成の増加を抑制する【JST・京大機械翻訳】

Author (5)： Ichise Taeko (Laboratory of Developmental Genetics, Center for Experimental Medicine and Systems Biology, The Institute of Medical Science, The University of Tokyo, Tokyo, Japan) ,  Ichise Taeko (Institute for Animal Research, Faculty of Medicine, University of the Ryukyus, Okinawa, Japan) ,  Yoshida Nobuaki (Laboratory of Developmental Genetics, Center for Experimental Medicine and Systems Biology, The Institute of Medical Science, The University of Tokyo, Tokyo, Japan) ,  Ichise Hirotake (Laboratory of Developmental Genetics, Center for Experimental Medicine and Systems Biology, The Institute of Medical Science, The University of Tokyo, Tokyo, Japan) ,  Ichise Hirotake (Institute for Animal Research, Faculty of Medicine, University of the Ryukyus, Okinawa, Japan)
Material： Journal of Pathology  (J Pathol)
Volume： 249  Issue： 1  Page： 39-51  Publication year： 2019 
JST Material Number： B0171A  ISSN： 0022-3417  CODEN： JPTLA  Document type： Article
Article type： 原著論文  Country of issue： United States (USA)  Language： ENGLISH (EN)

Abstract/Point： CREB-binding protein (CBP) and...

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Thesaurus term： *EGF receptor ,  *tumorigenesis ,  mutation ,  hyperplasia ,  transcription factor ,  *cellular signal transduction ,  carcinogenicity ,  keratinocyte ,  cell proliferation ,  *mouse(animal) ,  dysfunction ,  gene ,  *Ras protein ,  human(primates)
Semi thesaurus term： *p300 , 【Automatic Indexing@JST】

Author keywords (8)： CBP ,  p300 ,  Ras ,  Erk ,  EGFR ,  Mig6 ,  epidermis ,  hyperplasia

JST classification (4)： Chemistry,biochemistry and pathology of tumors(=neoplasms)  (GE02030N) ,  Biological function  (EB03040U) ,  Tumors(=neoplasms)of digestive system  (GH04000D) ,  Cell physiology in general  (EF02010S)

Terms in the title (10)： CBP ,  P300 ,  EGFR ,  Ras ,  ERK ,  シグナル伝達 ,  拮抗 ,  マウス ,  誘導 ,  腫瘍形成