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J-GLOBAL ID：202102230273699655   Reference number：21A0348682

SNAP23 deficiency causes severe brain dysplasia through the loss of radial glial cell polarity

SNAP23欠損は放射状グリア細胞極性の消失を介して重度の脳異形成を引き起こす【JST・京大機械翻訳】

Author (15)： Kunii Masataka (Department of Cell Biology, Graduate School of Medicine, Osaka University, Osaka, Japan) ,  Kunii Masataka (Laboratory of Molecular Traffic, Department of Molecular and Cellular Biology, Institute for Molecular and Cellular Regulation, Gunma University, Gunma, Japan) ,  Noguchi Yuria (Department of Cell Biology, Graduate School of Medicine, Osaka University, Osaka, Japan) ,  Yoshimura Shin-ichiro (Department of Cell Biology, Graduate School of Medicine, Osaka University, Osaka, Japan) ,  Kanda Satoshi (Department of Cell Biology, Graduate School of Medicine, Osaka University, Osaka, Japan) ,  Iwano Tomohiko (Department of Anatomy and Cell Biology, Graduate School of Medicine, University of Yamanashi, Yamanashi, Japan) ,  Avriyanti Erda (Department of Cell Biology, Graduate School of Medicine, Osaka University, Osaka, Japan) ,  Avriyanti Erda (Department of Dermatology and Venereology, Faculty of Medicine, Padjadjaran University, Bandung, Indonesia) ,  Atik Nur (Department of Cell Biology, Graduate School of Medicine, Osaka University, Osaka, Japan) ,  Atik Nur (Department of Biomedical Sciences, Faculty of Medicine, Padjadjaran University, Bandung, Indonesia) ,  Sato Takashi (Laboratory of Developmental Biology and Metabolism, Department of Molecular Medicine, Institute for Molecular and Cellular Regulation, Gunma University, Gunma, Japan) ,  Sato Ken (Laboratory of Molecular Traffic, Department of Molecular and Cellular Biology, Institute for Molecular and Cellular Regulation, Gunma University, Gunma, Japan) ,  Ogawa Masaharu (RIKEN Brain Science Institute, Saitama, Japan) ,  Harada Akihiro (Department of Cell Biology, Graduate School of Medicine, Osaka University, Osaka, Japan) ,  Harada Akihiro (Laboratory of Molecular Traffic, Department of Molecular and Cellular Biology, Institute for Molecular and Cellular Regulation, Gunma University, Gunma, Japan)
Material： Journal of Cell Biology  (JCB)
Volume： 220  Issue： 1  Page： Null  Publication year： 2020 
JST Material Number： C0575A  ISSN： 0021-9525  CODEN： JCLBA3  Document type： Article
Article type： 原著論文  Country of issue： United States (USA)  Language： ENGLISH (EN)

Abstract/Point： In the developing brain, the p...

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Thesaurus term： polarization (phenomenon) ,  Cerebellum ,  Proteins ,  Eumetopias jubata ,  complex (substance) ,  Cell Adhesion ,  *glia cell ,  ablation ,  Apoptosis ,  Neocortex ,  *Cell Polarity ,  N-cadherin ,  Neurogenesis ,  neural precursor ,  Brain Development ,  Brain ,  cell junction ,  binding and coupling

JST classification (1)： Function of cell components  (EF03060C)

Reference (64)：

• Assémat. Polarity complex proteins. Biochim. Biophys. Acta. 2008, 1778, 614. doi:10.1016/j.bbamem.2007.08.029
• Atik. The role of PKD in cell polarity, biosynthetic pathways, and organelle/F-actin distribution. Cell Struct. Funct. 2014, 39, 61. doi:10.1247/csf.13020
• Boncompain. Synchronization of secretory protein traffic in populations of cells. Nat. Methods. 2012, 9, 493. doi:10.1038/nmeth.1928
• Bryant. The ins and outs of E-cadherin trafficking. Trends Cell Biol. 2004, 14, 427. doi:10.1016/j.tcb.2004.07.007
• Bultje. Mammalian Par3 regulates progenitor cell asymmetric division via notch signaling in the developing neocortex. Neuron. 2009, 63, 189. doi:10.1016/j.neuron.2009.07.004

Terms in the title (5)： 欠損 ,  放射状 ,  グリア ,  細胞極性 ,  脳異形成