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J-GLOBAL ID:200902205649287166   Reference number:09A1277794

フィラデルフィア染色体陰性の慢性骨髄性白血病におけるBCR-ABL融合形成に伴う微小な染色体構造変化-複雑な再編成過程の考察-

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Material:
Volume: 10  Issue:Page: 351-359  Publication year: Nov. 30, 2009 
JST Material Number: L4223A  ISSN: 1347-2836  Document type: Article
Article type: 原著論文  Country of issue: Japan (JPN)  Language: JAPANESE (JA)
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Hematologic diseases 
Reference (16):
  • Richebourg S, et al: Mechanisms of genesis of variant translocation in chronic myeloid leukemia are not correlated with ABL1 or BCR deletion status or response to imatinib therapy. Cancer Genet Cytogenet 182: 95-102, 2008.
  • Gorusu M, et al: On the genesis and prognosis of variant translocations in chronic myeloid leukemia. Cancer Genet Cytogenet 173: 97-106, 2007.
  • 松原謙一、吉川 寛編: FISH実験プロトコール 秀潤社, 東京, 1994,
  • 保坂利江, 他: 派生9番染色体上の転座融合点領域欠失を伴う慢性骨髄性白血病3例のFISH法解析. 医学検査 56: 1125-1131, 2007.
  • De Melo VAS, et al: Deletions adjacent to BCR and ABL1 breakpoints occur in a substantial minority of chronic myeloid leukemia patients with masked Philadelphia rearrangements. Cancer Genet Cytogenet 182: 111-115, 2008.
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