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J-GLOBAL ID：201601018254305812 Update date: Apr. 01, 2025

Sato Yoshihumi

サトウヨシフミ | Sato Yoshihumi

Affiliation and department：
Job title： Assistant Professor

Research field (2)： Pathobiochemistry , Metabolism and endocrinology

Research keywords (5)：生化学 , 病態医科学 , 代謝学 , 老化 , Metabology

Research theme for competitive and other funds (20)：

2024 - 2026 膵β細胞低酸素の成因解明および制御法の検討
2023 - 2025 Elucidation of molecular mechanism of age-associated impaired insulin secretion by single cell analysis
2024 - 2025 インクレチンによるβ細胞酸素ホメオスタシス制御機構の解明
2022 - 2025 Regulation of glucose metabolism by SIRT7
2022 - 2024 The role of ANKS4B in glucose metabolism

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Papers (35)：

Mohammad Golam Kibria, Tatsuya Yoshizawa, Tianli Zhang, Katsuhiko Ono, Tomoya Mizumoto, Yoshifumi Sato, Tomohiro Sawa, Kazuya Yamagata. SIRT7 Is a Lysine Deacylase with a Preference for Depropionylation and Demyristoylation. International Journal of Molecular Sciences. 2025. 26. 7. 3153-3153
Kazuya Yamagata, Tomonori Tsuyama, Yoshifumi Sato. Roles of β-Cell Hypoxia in the Progression of Type 2 Diabetes. International Journal of Molecular Sciences. 2024. 25. 8. 4186-4186
Tomonori Tsuyama, Yoshifumi Sato, Tatsuya Yoshizawa, Takaaki Matsuoka, Kazuya Yamagata. Hypoxia causes pancreatic β-cell dysfunction and impairs insulin secretion by activating the transcriptional repressor BHLHE40. EMBO reports. 2023. e56227
Tatsuya Yoshizawa, Yoshifumi Sato, Shihab U Sobuz, Tomoya Mizumoto, Tomonori Tsuyama, Md Fazlul Karim, Keishi Miyata, Masayoshi Tasaki, Masaya Yamazaki, Yuichi Kariba, et al. SIRT7 suppresses energy expenditure and thermogenesis by regulating brown adipose tissue functions in mice. Nature communications. 2022. 13. 1. 7439-7439
Tomoya Mizumoto, Tatsuya Yoshizawa, Yoshifumi Sato, Takaaki Ito, Tomonori Tsuyama, Akiko Satoh, Satoshi Araki, Kenichi Tsujita, Masaru Tamura, Yuichi Oike, et al. SIRT7 Deficiency Protects against Aging-Associated Glucose Intolerance and Extends Lifespan in Male Mice. Cells. 2022. 11. 22

MISC (21)：

佐藤叔史, 津山友徳, 吉澤達也, 井上正宏, 山縣和也. 膵β細胞における低酸素誘導因子BHLHE40の機能解析. 生命科学系学会合同年次大会. 2017. 2017年度. [2P-1034]
佐藤叔史, 井上正宏, 吉澤達也, 山縣和也. β細胞低酸素による転写因子HNF-4α蛋白低下メカニズムの検討. 日本生化学会大会・日本分子生物学会年会合同大会講演要旨集. 2015. 88回・38回. [2P1214]-[2P1214]
佐藤叔史, 佐藤ちなみ, 吉澤達也, 井上正宏, 山縣和也. β細胞低酸素による転写因子HNF4α蛋白発現低下機序の検討. 糖尿病. 2015. 58. Suppl.1. S-442
佐藤叔史, 井上正宏, 山縣和也. 膵β細胞低酸素による転写因子発現低下メカニズムの検討. 糖尿病. 2014. 57. Suppl.1. S-191
芳川篤志, 今川彰久, 中田伸輔, 宇野彩, 福井健司, 黒田陽平, 宮田佑吾, 佐藤叔史, 花房俊昭, 松岡孝昭, et al. 劇症1型糖尿病患者膵島領域に発現する蛋白の同定(第三報) ISG15の機能解析. 糖尿病. 2014. 57. Suppl.1. S-129

Books (3)：

Lectures and oral presentations (33)：

HNF1α標的タンパク質TMED6によるβ細胞増殖調節機構の解明
(令和6年度日本生化学会九州支部例会)
膵β細胞における低酸素誘導性ATF3の役割
(第61回日本糖尿病学会九州地方会 2023)
Proinsulin and β-cell aging
(jds_65 2022)
転写因子HNF1αによる膵α細胞制御機構の解明
(第59回日本糖尿病学会九州地方会(九州支部賞受賞講演) 2021)
低酸素誘導性の転写抑制因子BHLHE40による膵β細胞障害機構
(第64 回日本糖尿病学会年次学術集会 2021)

Professional career (1)：

Work history (2)：

2011 - Kumamoto University Faculty of Life Sciences
2011 - Assistant Professor,Faculty of Life Sciences(Basic medicine group),Kumamoto University

Committee career (3)：

Awards (1)：

Association Membership(s) (3)：

THE MOLECULAR BIOLOGY SOCIETY OF JAPAN , THE JAPAN DIABETES SOCIETY , THE JAPANESE BIOCHEMICAL SOCIETY

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