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Rchr
J-GLOBAL ID：201801009059541884
Update date: Oct. 11, 2024 Kobashigawa Shinko
コバシガワ シンコ | Kobashigawa Shinko
ClipsLectures and oral presentations (25)
-
Involvement of mitochondrial ROS on DNA damage response through ATM activation after irradiation
(The 67th Annual Meeting of Japanese Radiation Research Society 2024) -
Mechanism of radiation-induced mutagenesis through the mitochondrial ROS generation
(The 66th Annual Meeting of Japanese Radiation Research Society 2023) -
Mitochondria-derived ROS sustain ATM activation and participate in radiation-induced cellular senescence
(The 19th Ataxia-Telangiectasia workshop (ATW2023Kyoto) 2023) -
Involvement mechanism of mitochondrial-derived delayed reactive oxygen species as a protective target for normal cells in radiotherapy
(2023) -
Contribution to delayed ROS generation and induction of cellular senescence by carbon beam irradiation
(2022) -
Mitochondrial ROS by ionizing radiation induce cellular senescence throught ATM activation
(Japan Radiation Society 2021) -
Delayed reactive oxygen species contribute to radiation-induced cellular senescence
(Japan Radiation Society 2020) -
Radioprotective effect by post-irradiation treatment with glucopyranosyl ascorbic acid
(2020) -
Different response to radiation-induced delayed oxidative stress in normal cells and cancer cells
(The 8th GIAR International Symposium 2020) -
Analysis of cell secretory factors that alter the radiosensitivity
(Japan Radiation Society 2019) -
Difference of normal cells and cancer cells for response of radiation induced delayed oxidative stress
(Japan Radiation Society 2019) -
Delayed ROS related to mitochondrial fission causes cellular senescence after radiation exposure.
(ASUKA Symposium 2019 2019) -
グルコピラノシルアスコルビン酸の照射後処理による放射線感受性の変化 ~正常細胞 とがん細胞の比較~
(日本放射線影響学会第61回大会 2018) -
放射線による細胞老化は遅発性活性酸素種が関与する
(日本放射線影響学会第60回大会 2017) -
放射線による不可逆的老化様細胞増殖停止への遅発性活性酸素種の関与
(第46回放射線による制癌シンポジウム・第55回生物部会学術大会 2017) -
培養上清成分による放射線感受性の変化
(Japanese Radiation Research Society 2015) -
Radiation induced delayed oxidative stress is related to p53 dependent senescence-like-growth arrest.
(15th International Congress of Radiation Research. 2015) -
Analysis of the biological effects of 18F at different intracellular levels.
(15th International Congress of Radiation Research. 2015) -
癌細胞のバイスタンダー効果を介した放射線抵抗性獲得機構
(日本放射線影響学会 第57回大会 2014) -
放射線誘発遅発性活性酸素種は細胞死を引き起こすか?
(日本放射線影響学会第57回大会 2014)
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