文献

J-GLOBAL ID：201602228175530141   整理番号：16A1175947

IL-17A-産生CD30⁺Vδ1 T細胞は炎症誘発性の癌の進行を促す

IL-17A-producing CD30⁺ Vδ1 T cells drive inflammation-induced cancer progression

著者 (15件)： Kimura Yoshitaka (Laboratory of Cancer Biology and Molecular Immunology, Graduate School of Pharmaceutical Sciences, University of Tokyo, Bunkyo-ku, Tokyo, Japan) ,  Nagai Nao (Laboratory of Cancer Biology and Molecular Immunology, Graduate School of Pharmaceutical Sciences, University of Tokyo, Bunkyo-ku, Tokyo, Japan) ,  Tsunekawa Naoki (Laboratory of Cancer Biology and Molecular Immunology, Graduate School of Pharmaceutical Sciences, University of Tokyo, Bunkyo-ku, Tokyo, Japan) ,  Sato-Matsushita Marimo (Department of Surgery and Bioengineering, Institute of Medical Science, University of Tokyo, Minato-ku, Tokyo, Japan) ,  Yoshimoto Takayuki (Department of Immunoregulation, Institute of Medical Science, Tokyo Medical University, Shinjuku-ku, Tokyo, Japan) ,  Cua Daniel J. (Pathway Biology, Merck Research Laboratories, Palo Alto, California, USA) ,  Iwakura Yoichiro (Center for Animal Disease Models, Research Institute for Biomedical Sciences, Tokyo University of Science, Noda, Chiba, Japan) ,  Yagita Hideo (Department of Immunology, Juntendo University School of Medicine, Bunkyo-ku, Tokyo, Japan) ,  Okada Futoshi (Division of Pathological Biochemistry, Tottori University Faculty of Medicine, Yonago, Tottori, Japan) ,  Okada Futoshi (Chromosome Engineering Research Center, Tottori University, Yonago, Tottori, Japan) ,  Tahara Hideaki (Department of Surgery and Bioengineering, Institute of Medical Science, University of Tokyo, Minato-ku, Tokyo, Japan) ,  Saiki Ikuo (Division of Pathogenic Biochemistry, Department of Bioscience, Institute of Natural Medicine, University of Toyama, Toyama, Toyama, Japan) ,  Irimura Tatsuro (Laboratory of Cancer Biology and Molecular Immunology, Graduate School of Pharmaceutical Sciences, University of Tokyo, Bunkyo-ku, Tokyo, Japan) ,  Hayakawa Yoshihiro (Laboratory of Cancer Biology and Molecular Immunology, Graduate School of Pharmaceutical Sciences, University of Tokyo, Bunkyo-ku, Tokyo, Japan. [e-mail]) ,  Hayakawa Yoshihiro (Division of Pathogenic Biochemistry, Department of Bioscience, Institute of Natural Medicine, University of Toyama, Toyama, Toyama, Japan. [e-mail])
資料名： Cancer Science (Web)  (Cancer Science (Web))
巻： 107  号： 9  ページ： 1206-1214  発行年： 2016年 
JST資料番号： U0505A  ISSN： 1349-7006  資料種別： 逐次刊行物 (A)
記事区分： 原著論文  発行国： アメリカ合衆国 (USA)  言語： 英語 (EN)

抄録/ポイント： 炎症は高い腫瘍転移性と関係することが疑われているが,癌の進行...

シソーラス用語： *CD抗原 ,...
準シソーラス用語： *CD30 ,...

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著者キーワード (5件)： CD30 ,  IL-17 ,  IL-1β ,  neutrophil ,  γδ T cell

分類 (1件)： 腫ようの化学・生化学・病理学  (GE02030N)

引用文献 (41件)：

• Hanahan D, Weinberg RA. Hallmarks of cancer: The next generation. Cell 2011; 144: 646-74.
• Grivennikov SI, Greten FR, Karin M. Immunity, inflammation, and cancer. Cell 2010; 140: 883 -99.
• Elinav E, Nowarski R, Thaiss CA, Hu B, Jin C, Flavell RA. Inflammation-induced cancer: Crosstalk between tumours, immune cells and microorganisms. Nat Rev Cancer 2013; 13: 759-71.
• Iwakura Y, Ishigame H, Saijo S, Nakae S. Functional specialization of interleukin-17 family members. Immunity 2011; 34: 149-62.
• Miossec P, Kolls JK. Targeting IL-17 and TH17 cells in chronic inflammation. Nat Rev Drug Discov 2012; 11: 763-76.

タイトルに関連する用語 (4件)： IL-17A ,  T細胞 ,  炎症 ,  癌